Literature DB >> 28341273

Branched-chain amino acids enhance cyst development in autosomal dominant polycystic kidney disease.

Junya Yamamoto1, Saori Nishio2, Fumihiko Hattanda1, Daigo Nakazawa1, Toru Kimura3, Michio Sata4, Minoru Makita1, Yasunobu Ishikawa1, Tatsuya Atsumi1.   

Abstract

Autosomal dominant polycystic kidney disease (ADPKD) is characterized by the progressive development of kidney and liver cysts. The mammalian target of rapamycin (mTOR) cascade is one of the important pathways regulating cyst growth in ADPKD. Branched-chain amino acids (BCAAs), including leucine, play a crucial role to activate mTOR pathway. Therefore, we administered BCAA dissolved in the drinking water to Pkd1flox/flox:Mx1-Cre (cystic) mice from four to 22 weeks of age after polyinosinic-polycytidylic acid-induced conditional Pkd1 knockout at two weeks of age. The BCAA group showed significantly greater kidney/body weight ratio and higher cystic index in both the kidney and liver compared to the placebo-treated mice. We found that the L-type amino acid transporter 1 that facilitates BCAA entry into cells is strongly expressed in cells lining the cysts. We also found increased cyst-lining cell proliferation and upregulation of mTOR and mitogen-activated protein kinase/extracellular signal-regulated kinase (MAPK/ERK) pathways in the BCAA group. In vitro, we cultured renal epithelial cell lines from Pkd1 null mice with or without leucine. Leucine was found to stimulate cell proliferation, as well as activate mTOR and MAPK/ERK pathways in these cells. Thus, BCAA accelerated disease progression by mTOR and MAPK/ERK pathways. Hence, BCAA may be harmful to patients with ADPKD.
Copyright © 2017 International Society of Nephrology. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  ADPKD; BCAA; LAT-1; mTOR

Mesh:

Substances:

Year:  2017        PMID: 28341273     DOI: 10.1016/j.kint.2017.01.021

Source DB:  PubMed          Journal:  Kidney Int        ISSN: 0085-2538            Impact factor:   10.612


  6 in total

1.  Hydrochlorothiazide ameliorates polyuria caused by tolvaptan treatment of polycystic kidney disease in PCK rats.

Authors:  Anyi Wang; Takuo Hirose; Yusuke Ohsaki; Chika Takahashi; Emiko Sato; Ikuko Oba-Yabana; Satoshi Kinugasa; Yoshikazu Muroya; Sadayoshi Ito; Takefumi Mori
Journal:  Clin Exp Nephrol       Date:  2018-11-13       Impact factor: 2.801

2.  Dietary Interventions in Autosomal Dominant Polycystic Kidney Disease.

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Review 3.  Molecular Mechanisms of Epigenetic Regulation, Inflammation, and Cell Death in ADPKD.

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Journal:  Front Mol Biosci       Date:  2022-06-29

Review 4.  Nutritional therapy in autosomal dominant polycystic kidney disease.

Authors:  Biagio R Di Iorio; Adamasco Cupisti; Claudia D'Alessandro; Antonio Bellasi; Vincenzo Barbera; Luca Di Lullo
Journal:  J Nephrol       Date:  2018-01-17       Impact factor: 3.902

5.  Krüppel-like factor 6-mediated loss of BCAA catabolism contributes to kidney injury in mice and humans.

Authors:  Sian E Piret; Yiqing Guo; Ahmed A Attallah; Sylvia J Horne; Amy Zollman; Daniel Owusu; Justina Henein; Viktoriya S Sidorenko; Monica P Revelo; Takashi Hato; Avi Ma'ayan; John Cijiang He; Sandeep K Mallipattu
Journal:  Proc Natl Acad Sci U S A       Date:  2021-06-08       Impact factor: 11.205

6.  Metabolic Phenotyping of Anks3 Depletion in mIMCD-3 cells - a Putative Nephronophthisis Candidate.

Authors:  Manuel Schlimpert; Simon Lagies; Vadym Budnyk; Barbara Müller; Gerd Walz; Bernd Kammerer
Journal:  Sci Rep       Date:  2018-06-13       Impact factor: 4.379

  6 in total

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