Literature DB >> 28336108

Vitamin D increases PPARγ expression and promotes beneficial effects of physical activity in metabolic syndrome.

Rastegar Hoseini1, Arsalan Damirchi1, Parvin Babaei2.   

Abstract

OBJECTIVES: Peroxisome proliferator-activated receptor (PPAR) and vitamin D signaling pathways regulate a multitude of genes involved in different physiological functions. The aim of the present study was to examine the effects of vitamin D supplementation and aerobic training on metabolic syndrome and PPARγ expression.
METHODS: Forty female ovariectomized rats were divided into five groups of aerobic training with high (OVX + Exe + HD), moderate (OVX + Exe + MD), and low dose of vitamin D (OVX + Exe + LD), aerobic training receving vehicle (sesame oil; OVX + Exe + oil), and sham-operated control (sham) groups. After 2 mo of treatment, serum insulin, vitamin D, glucose, lipid profile, visceral fat, and liver PPARγ gene expression were measured.
RESULTS: The combination of exercise and high doses of vitamin D significantly reduced insulin (P = 0.039), blood glucose (P = 0.024), and homeostatic model assessment for insulin resistance (P = 0.011), and elevated PPARγ gene expression (P = 0.032). Also, treatment with aerobic training and either high or moderate vitamin D, ameliorated overall metabolic syndrome Z scores (P = 0.001).
CONCLUSION: Findings from the present study suggested that a sedentary lifestyle and vitamin D deficiency accelerated the occurrence of metabolic syndrome probably by decreasing the expression of nuclear receptor PPARγ. Additionally, adequate levels of plasma vitamin D are necessary to achieve the beneficial metabolic effects of physical activity.
Copyright © 2016 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Exercise; Metabolic syndrome; PPARγ; Vitamin D

Mesh:

Substances:

Year:  2016        PMID: 28336108     DOI: 10.1016/j.nut.2016.06.010

Source DB:  PubMed          Journal:  Nutrition        ISSN: 0899-9007            Impact factor:   4.008


  14 in total

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Review 10.  Understanding the Role of Exercise in Nonalcoholic Fatty Liver Disease: ERS-Linked Molecular Pathways.

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