Literature DB >> 28279569

Lifetime exercise intolerance with lactic acidosis as key manifestation of novel compound heterozygous ACAD9 mutations causing complex I deficiency.

Bertold Schrank1, Benedikt Schoser2, Thomas Klopstock3, Peter Schneiderat2, Rita Horvath4, Angela Abicht5, Elke Holinski-Feder5, Sarunas Augustis6.   

Abstract

We report a 36-year-old female having lifetime exercise intolerance and lactic acidosis with nausea associated with novel compound heterozygous Acyl-CoA dehydrogenase 9 gene (ACAD9) mutations (p.Ala390Thr and p.Arg518Cys). ACAD9 is an assembly factor for the mitochondrial respiratory chain complex I. ACAD9 mutations are recognized as frequent causes of complex I deficiency. Our patient presented with exercise intolerance, rapid fatigue, and nausea since early childhood. Mild physical workload provoked the occurrence of nausea and vomiting repeatedly. Her neurological examination, laboratory findings and muscle biopsy demonstrated no abnormalities. A bicycle spiroergometry provoked significant lactic acidosis during and following exercise pointing towards a mitochondrial disorder. Subsequently, the analysis of respiratory chain enzyme activities in muscle revealed severe isolated complex I deficiency. Candidate gene sequencing revealed two novel heterozygous ACAD9 mutations. This patient report expands the mutational and phenotypic spectrum of diseases associated with mutations in ACAD9.
Copyright © 2017 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  ACAD9 mutations; Complex I deficiency; Isolated myopathy; Life-long exercise intolerance

Mesh:

Substances:

Year:  2017        PMID: 28279569     DOI: 10.1016/j.nmd.2017.02.005

Source DB:  PubMed          Journal:  Neuromuscul Disord        ISSN: 0960-8966            Impact factor:   4.296


  3 in total

1.  Mitochondrial multiorgan disorder syndrome (MIMODS) due to a compound heterozygous mutation in the ACAD9 gene.

Authors:  Josef Finsterer; Sinda Zarrouk-Mahjoub
Journal:  Mol Genet Metab Rep       Date:  2017-07-28

2.  Role of lactic acidosis as a mediator of sprint-mediated nausea.

Authors:  Robert J Merrells; Ashley J Cripps; Paola T Chivers; Paul A Fournier
Journal:  Physiol Rep       Date:  2019-11

Review 3.  Human diseases associated with defects in assembly of OXPHOS complexes.

Authors:  Daniele Ghezzi; Massimo Zeviani
Journal:  Essays Biochem       Date:  2018-07-20       Impact factor: 8.000

  3 in total

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