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Literature DB >> 28279333

DAI Another Way: Necroptotic Control of Viral Infection.

Abstract

Interrogation of murine cytomegalovirus (MCMV)-encoded cell-death suppressors revealed that necroptosis functions as a trap door to eliminate virally infected cells. This crucial host defense pathway is orchestrated by the sensing of infection by DAI/ZBP-1, engagement of the kinase RIPK3, and subsequent membrane permeablization by the pseudokinase MLKL.

Entities: Gene Species

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Year: 2017 PMID： 28279333 DOI： 10.1016/j.chom.2017.01.016

Source DB: PubMed Journal: Cell Host Microbe ISSN： 1931-3128 Impact factor: 21.023

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Cited

12 in total

1. Direct Activation of Human MLKL by a Select Repertoire of Inositol Phosphate Metabolites.

Authors: Dan E McNamara; Cole M Dovey; Andrew T Hale; Giovanni Quarato; Christy R Grace; Cristina D Guibao; Jonathan Diep; Amanda Nourse; Casey R Cai; Hong Wu; Ravi C Kalathur; Douglas R Green; John D York; Jan E Carette; Tudor Moldoveanu
Journal: Cell Chem Biol Date: 2019-04-25 Impact factor: 8.116

2. Characterization of MLKL-mediated Plasma Membrane Rupture in Necroptosis.

Authors: Dan E McNamara; Giovanni Quarato; Cliff S Guy; Douglas R Green; Tudor Moldoveanu
Journal: J Vis Exp Date: 2018-08-07 Impact factor: 1.355

3. TRADD mediates the tumor necrosis factor-induced apoptosis of L929 cells in the absence of RIP3.

Authors: Xixi Chang; Lili Wang; Zicheng Wang; Shuai Wu; Xiaoming Zhu; Shiping Hu; Yu Wang; Jiyun Yu; Guozhu Chen
Journal: Sci Rep Date: 2017-11-23 Impact factor: 4.379

4. RIP1 kinase activity is critical for skin inflammation but not for viral propagation.

Authors: Joshua D Webster; Youngsu C Kwon; Summer Park; Hua Zhang; Nick Corr; Nina Ljumanovic; Adeyemi O Adedeji; Eugene Varfolomeev; Tatiana Goncharov; Jessica Preston; Sara F Santagostino; Snahel Patel; Min Xu; Jonathan Maher; Brent S McKenzie; Domagoj Vucic
Journal: J Leukoc Biol Date: 2020-01-27 Impact factor: 4.962

10. Evolutionary Profile for (Host and Viral) MLKL Indicates Its Activities as a Battlefront for Extensive Counteradaptation.

Authors: Suzette N Palmer; Sruthi Chappidi; Chelsea Pinkham; Dustin C Hancks
Journal: Mol Biol Evol Date: 2021-12-09 Impact factor: 16.240

DAI Another Way: Necroptotic Control of Viral Infection.

1. Direct Activation of Human MLKL by a Select Repertoire of Inositol Phosphate Metabolites.

2. Characterization of MLKL-mediated Plasma Membrane Rupture in Necroptosis.

3. TRADD mediates the tumor necrosis factor-induced apoptosis of L929 cells in the absence of RIP3.

4. RIP1 kinase activity is critical for skin inflammation but not for viral propagation.

Review 5. Pathways of host cell exit by intracellular pathogens.

6. Necroptosis mediators RIPK3 and MLKL suppress intracellular Listeria replication independently of host cell killing.

7. TRADD Mediates RIPK1-Independent Necroptosis Induced by Tumor Necrosis Factor.

8. USP22 controls necroptosis by regulating receptor-interacting protein kinase 3 ubiquitination.

Review 9. MLKL: Functions beyond serving as the Executioner of Necroptosis.

10. Evolutionary Profile for (Host and Viral) MLKL Indicates Its Activities as a Battlefront for Extensive Counteradaptation.