Erratum to: Specific disruption of Lnk in murine endothelial progenitor cells promotes dermal wound healing via enhanced vasculogenesis, activation of myofibroblasts, and suppression of inflammatory cell recruitment.

Erratum

Unfortunately, after publication of this article [1], it was noticed that Fig. 4 was incorrect. Panels B and D contained incorrect graphs. The corrected Fig. 4 can be seen below and the original article has been updated to correct this.

Fig. 4

A transplant of Lnk-deficient EPCs suppresses the recruitment of inflammatory cells. After injection of wild-type (WT) and Lnk-deficient EPCs into wound sites, wound tissues were analyzed to determine the recruitment of cytotoxic T cells (CD3- and CD8-positive cells), macrophages (CD11b-positive cells), and neutrophils (CD45-positive cells) on postoperative days 3 and 7. a The recruitment of cytotoxic T cells in wound tissues was assessed by FACS analysis. b The percentage of CD3/CD8 double-positive cells on postoperative days 3 and 7. Values are mean ± SEM; **p < 0.01 compared to postoperative day 3, respectively, and ##p < 0.01 compared to injection with WT EPCs. c The recruitment of macrophages and neutrophils to wound tissues was assessed by FACS analysis. d The percentage of CD11b- and CD45-positive cells on postoperative days 3 and 7. Values are mean ± SEM;**p < 0.01 compared to postoperative day 3, respectively, #p < 0.05 and ##p < 0.01 compared to injection with WT EPCs]