Literature DB >> 28232325

Alk2/ACVR1 and Alk3/BMPR1A Provide Essential Function for Bone Morphogenetic Protein-Induced Retinal Angiogenesis.

Heon-Woo Lee1, Diana C Chong1, Roxana Ola1, William P Dunworth1, Stryder Meadows1, Jun Ka1, Vesa M Kaartinen1, Yibing Qyang1, Ondine Cleaver1, Victoria L Bautch2, Anne Eichmann1, Suk-Won Jin1.   

Abstract

OBJECTIVE: Increasing evidence suggests that bone morphogenetic protein (BMP) signaling regulates angiogenesis. Here, we aimed to define the function of BMP receptors in regulating early postnatal angiogenesis by analysis of inducible, endothelial-specific deletion of the BMP receptor components Bmpr2 (BMP type 2 receptor), Alk1 (activin receptor-like kinase 1), Alk2, and Alk3 in mouse retinal vessels. APPROACH AND
RESULTS: Expression analysis of several BMP ligands showed that proangiogenic BMP ligands are highly expressed in postnatal retinas. Consistently, BMP receptors are also strongly expressed in retina with a distinct pattern. To assess the function of BMP signaling in retinal angiogenesis, we first generated mice carrying an endothelial-specific inducible deletion of Bmpr2. Postnatal deletion of Bmpr2 in endothelial cells substantially decreased the number of angiogenic sprouts at the vascular front and branch points behind the front, leading to attenuated radial expansion. To identify critical BMPR1s (BMP type 1 receptors) associated with BMPR2 in retinal angiogenesis, we generated endothelial-specific inducible deletion of 3 BMPR1s abundantly expressed in endothelial cells and analyzed the respective phenotypes. Among these, endothelial-specific deletion of either Alk2/acvr1 or Alk3/Bmpr1a caused a delay in radial expansion, reminiscent of vascular defects associated with postnatal endothelial-specific deletion of BMPR2, suggesting that ALK2/ACVR1 and ALK3/BMPR1A are likely to be the critical BMPR1s necessary for proangiogenic BMP signaling in retinal vessels.
CONCLUSIONS: Our data identify BMP signaling mediated by coordination of ALK2/ACVR1, ALK3/BMPR1A, and BMPR2 as an essential proangiogenic cue for retinal vessels.
© 2017 The Authors.

Entities:  

Keywords:  BMP signaling; angiogenesis; retina; vertebrate development

Mesh:

Substances:

Year:  2017        PMID: 28232325      PMCID: PMC5382795          DOI: 10.1161/ATVBAHA.116.308422

Source DB:  PubMed          Journal:  Arterioscler Thromb Vasc Biol        ISSN: 1079-5642            Impact factor:   8.311


  30 in total

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3.  Identification of BMP9 and BMP10 as functional activators of the orphan activin receptor-like kinase 1 (ALK1) in endothelial cells.

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Journal:  Blood       Date:  2006-10-26       Impact factor: 22.113

4.  Specificity and structure of a high affinity activin receptor-like kinase 1 (ALK1) signaling complex.

Authors:  Sharon A Townson; Erik Martinez-Hackert; Chloe Greppi; Patricia Lowden; Dianne Sako; June Liu; Jeffrey A Ucran; Katia Liharska; Kathryn W Underwood; Jasbir Seehra; Ravindra Kumar; Asya V Grinberg
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5.  Multiple roles for activin-like kinase-2 signaling during mouse embryogenesis.

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8.  The type I serine/threonine kinase receptor ActRIA (ALK2) is required for gastrulation of the mouse embryo.

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Authors:  David M Wiley; Jun-Dae Kim; Jijun Hao; Charles C Hong; Victoria L Bautch; Suk-Won Jin
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10.  Alk1 and Alk5 inhibition by Nrp1 controls vascular sprouting downstream of Notch.

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  21 in total

1.  Developmental SMAD6 loss leads to blood vessel hemorrhage and disrupted endothelial cell junctions.

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2.  Cerebral Vein Malformations Result from Loss of Twist1 Expression and BMP Signaling from Skull Progenitor Cells and Dura.

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Review 3.  Vascular Development.

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6.  Endoglin moves and shapes endothelial cells.

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Journal:  Nat Cell Biol       Date:  2017-05-31       Impact factor: 28.824

7.  Reporting Sex and Sex Differences in Preclinical Studies.

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9.  Transcriptomic analysis identifies novel targets for individual bone morphogenetic protein type 1 receptors in endothelial cells.

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Review 10.  Consequences of BMPR2 Deficiency in the Pulmonary Vasculature and Beyond: Contributions to Pulmonary Arterial Hypertension.

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Journal:  Int J Mol Sci       Date:  2018-08-24       Impact factor: 5.923

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