Literature DB >> 28213001

Predictive factors for EGFR-tyrosine kinase inhibitor retreatment in patients with EGFR-mutated non-small-cell lung cancer - A multicenter retrospective SEQUENCE study.

Gee-Chen Chang1, Chien-Hua Tseng2, Kuo-Hsuan Hsu3, Chong-Jen Yu4, Cheng-Ta Yang5, Kun-Chieh Chen6, Tsung-Ying Yang7, Jeng-Sen Tseng7, Chien-Ying Liu4, Wei-Yu Liao5, Te-Chun Hsia8, Chih-Yen Tu9, Meng-Chih Lin10, Ying-Huang Tsai11, Meng-Jer Hsieh12, Wen-Shuo Wu13, Yuh-Min Chen14.   

Abstract

BACKGROUND: Acquired resistance occurs in most non-small cell lung cancer (NSCLC) patients with epidermal growth factor receptor (EGFR) mutations experiencing a response to EGFR-tyrosine kinase inhibitor (TKI) initially. We investigated EGFR-TKI retreatment in patients who had previously received EGFR-TKI followed by chemotherapy.
MATERIALS AND METHODS: This was a retrospective multicenter study. Patients with locally advanced or metastatic adenocarcinoma or TTF-1 (+) NSCLC, positive EGFR sensitive mutation, and EGFR-TKI reuse after initial EGFR-TKI followed by chemotherapy were enrolled. The objectives were to assess the objective response rate (ORR), disease control rate (DCR), progression-free survival (PFS), and overall survival (OS) of EGFR TKI switched retreatment.
RESULTS: In total, 205 patients were enrolled, with a median age of 61.8 years (range 31.4-92.9). There was a larger proportion of females (62.9%) than males, and more never-smokers (73.2%) than ever-smokers. In the initial EGFR-TKI administration, 57.6% of patients showed a complete response (CR) or partial response (PR), and 34.6% had stable disease (SD); in the second-line chemotherapy, 13.7% had PR, and 58.0% had SD; in the EGFR-TKI retreatment, 7.3% had PR, and 37.1% had SD. The median PFS of first-line EGFR-TKI was 8.0 months (95% CI 7.3-8.2), and retreatment EGFR-TKI was 4.1 months (95% CI 2.7-4.6). The median OS since the start of the first-line EGFR-TKI therapy was 35.9 months (95% CI 28.8-50.9), and since the start of EGFR-TKI retreatment was 12.6 months (95% CI 10.4-20.9). In the univariable and multivariable regression analysis of factors associated with PFS of EGFR-TKI retreatment, time interval between the two EGFR TKIs equal to or more than 7 months was statistically significant (HR=0.62, 95% CI 0.44-0.86; HR=0.6, 95% CI 0.43-0.86), both p<0.01. Females with exon 21 mutation also showed a significant difference between the two groups (HR=0.51, 95% CI 0.30-0.86; HR=0.52 (0.31-0.88), both p<0.05).
CONCLUSIONS: EGFR-TKI retreatment was effective in prolonging survival, and was shown to be a worthwhile option for EGFR-mutated NSCLC patients after failure of first-line EGFR-TKI and chemotherapy. The survival benefit was especially pronounced in patients with longer drug holidays from the initial EGFR-TKI and in females with the exon 21 mutation. Copyright Â
© 2016 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  Adenocarcinoma; Drug holiday; EGFR TKI retreatment; EGFR mutation; Exon 21 mutation; Females; Lung cancer; Overall survival; Progression free survival

Mesh:

Substances:

Year:  2016        PMID: 28213001     DOI: 10.1016/j.lungcan.2016.12.002

Source DB:  PubMed          Journal:  Lung Cancer        ISSN: 0169-5002            Impact factor:   5.705


  10 in total

Review 1.  State-of-the-Art Molecular Oncology of Lung Cancer in Taiwan.

Authors:  Yung-Hung Luo; Kung-Hao Liang; Hsu-Ching Huang; Chia-I Shen; Chi-Lu Chiang; Mong-Lien Wang; Shih-Hwa Chiou; Yuh-Min Chen
Journal:  Int J Mol Sci       Date:  2022-06-24       Impact factor: 6.208

2.  Heterogeneity and Coexistence of T790M and T790 Wild-Type Resistant Subclones Drive Mixed Response to Third-Generation Epidermal Growth Factor Receptor Inhibitors in Lung Cancer.

Authors:  Zofia Piotrowska; Mehlika Hazar-Rethinam; Coleen Rizzo; Brandon Nadres; Emily E Van Seventer; Heather A Shahzade; Inga T Lennes; Anthony J Iafrate; Dora Dias-Santagata; Ignaty Leshchiner; Nicholas A Jessop; Haichuan Hu; Subba R Digumarthy; Rebecca J Nagy; Richard B Lanman; Susan Moody; Matthew J Niederst; Jeffrey A Engelman; Aaron N Hata; Ryan B Corcoran; Lecia V Sequist
Journal:  JCO Precis Oncol       Date:  2018-07-16

3.  Host-Dependent Phenotypic Resistance to EGFR Tyrosine Kinase Inhibitors.

Authors:  Yuya Haga; Ilaria Marrocco; Ashish Noronha; Mary Luz Uribe; Nishanth Belugali Nataraj; Arunachalam Sekar; Diana Drago-Garcia; Simone Borgoni; Moshit Lindzen; Suvendu Giri; Stefan Wiemann; Yasuo Tsutsumi; Yosef Yarden
Journal:  Cancer Res       Date:  2021-05-03       Impact factor: 12.701

4.  Reverse epithelial-mesenchymal transition contributes to the regain of drug sensitivity in tyrosine kinase inhibitor-resistant non-small cell lung cancer cells.

Authors:  An-Fu Lee; Man-Chin Chen; Chao-Ju Chen; Chih-Jen Yang; Ming-Shyang Huang; Yu-Peng Liu
Journal:  PLoS One       Date:  2017-07-06       Impact factor: 3.240

5.  A novel EGFR-TKI inhibitor (cAMP-H3BO3complex) combined with thermal therapy is a promising strategy to improve lung cancer treatment outcomes.

Authors:  Yongpeng Tong; Chunliu Huang; Junfang Zhang
Journal:  Oncotarget       Date:  2017-05-05

6.  Circulating mutational portrait of cancer: manifestation of aggressive clonal events in both early and late stages.

Authors:  Meng Yang; Umit Topaloglu; W Jeffrey Petty; Matthew Pagni; Kristie L Foley; Stefan C Grant; Mac Robinson; Rhonda L Bitting; Alexandra Thomas; Angela T Alistar; Rodwige J Desnoyers; Michael Goodman; Carol Albright; Mercedes Porosnicu; Mihaela Vatca; Shadi A Qasem; Barry DeYoung; Ville Kytola; Matti Nykter; Kexin Chen; Edward A Levine; Edgar D Staren; Ralph B D'Agostino; Robin M Petro; William Blackstock; Bayard L Powell; Edward Abraham; Boris Pasche; Wei Zhang
Journal:  J Hematol Oncol       Date:  2017-05-04       Impact factor: 17.388

7.  Intercalated Treatment Following Rebiopsy Is Associated with a Shorter Progression-Free Survival of Osimertinib Treatment.

Authors:  Jeng-Sen Tseng; Tsung-Ying Yang; Kun-Chieh Chen; Kuo-Hsuan Hsu; Yen-Hsiang Huang; Kang-Yi Su; Sung-Liang Yu; Gee-Chen Chang
Journal:  Cancer Res Treat       Date:  2017-12-11       Impact factor: 4.679

8.  Knockdown of CENPF inhibits the progression of lung adenocarcinoma mediated by ERβ2/5 pathway.

Authors:  Tang Hexiao; Bai Yuquan; Xiong Lecai; Wei Yanhong; Shen Li; Hu Weidong; Xu Ming; Zhou Xuefeng; Pan Gaofeng; Zhang Li; Zhu Minglin; Tang Zheng; Yang Zetian; Zhou Xiao; Cai Yi; Michael Lanuti; Zhao Jinping
Journal:  Aging (Albany NY)       Date:  2021-01-10       Impact factor: 5.682

9.  The miR-3127-5p/p-STAT3 axis up-regulates PD-L1 inducing chemoresistance in non-small-cell lung cancer.

Authors:  Dongfang Tang; Dandan Zhao; Yun Wu; Ruyong Yao; Lin Zhou; Liming Lu; Wen Gao; Yifeng Sun
Journal:  J Cell Mol Med       Date:  2018-05-04       Impact factor: 5.295

10.  CaSR Induces Osteoclast Differentiation and Promotes Bone Metastasis in Lung Adenocarcinoma.

Authors:  Lian Liu; Yichang Fan; Zhaoxin Chen; Yujian Zhang; Jing Yu
Journal:  Front Oncol       Date:  2020-03-25       Impact factor: 6.244

  10 in total

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