Literature DB >> 28209096

Increased oxidative stress alters nucleosides metabolite levels in sickle cell anemia.

Lívia Gelain Castilhos1, Juliana Sorraila de Oliveira2, Stephen Adeniyi Adefegha2,3, Luana Pereira Magni2, Pedro Henrique Doleski2, Fatima Husein Abdalla2, Cínthia Melazzo de Andrade2, Daniela Bitencourt Rosa Leal1,2.   

Abstract

OBJECTIVES: This study was conducted to assess the markers of oxidative stress, myeloperoxidase (MPO), acetylcholinesterase (AChE) and xanthine oxidase (XO) activities as well as the levels of nucleotide metabolites in sickle cell anemia (SCA) patients.
METHODS: Fifteen SCA treated patients and 30 health subjects (control group) were selected. The markers of oxidative stress (levels of reactive oxygen species (ROS), plasma proteins, carbonyl content, lipid peroxidation (TBARS), total thiols (T-SH), glutathione and catalase activity), MPO, AChE and XO activities as well as the levels of nucleotide metabolites were measured in SCA patients.
RESULTS: ROS, thiobarbituric acid-reactive substances (TBARS) and T-SH levels as well as the activities of catalase and MPO were significantly increased while glutathione level was reduced in SCA patients. Furthermore, a significant (P < 0.001) increase in hypoxanthine level was demonstrated in SCA patients. However, the serum levels for xanthine (P < 0.01) and uric acid (P < 0.001) were decreased in SCA patients. A significant (P < 0.001) decrease in XO activity was detected in SCA patients. DISCUSSION: The altered parameters in SCA patients suggest that the generation and impairment of oxidative stress in this disease as well as antioxidant markers are contributory factors towards cellular redox homeostasis and alteration of purine metabolites.

Entities:  

Keywords:  Sickle cell anemia; enzymes; hypoxanthine; lipid peroxidation; oxidative stress; reactive oxygen species; uric acid; xanthine

Mesh:

Substances:

Year:  2017        PMID: 28209096      PMCID: PMC6837566          DOI: 10.1080/13510002.2017.1288973

Source DB:  PubMed          Journal:  Redox Rep        ISSN: 1351-0002            Impact factor:   4.412


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