Literature DB >> 28197741

Correlation between the COL4A3, MMP-9, and TIMP-1 polymorphisms and risk of keratoconus.

Ramin Saravani1,2, Davood Yari3,4, Samira Saravani5, Farzaneh Hasanian-Langroudi2.   

Abstract

PURPOSE: Keratoconus (KC) is thinning of the central cornea. Its etiology is unknown, but it may result from degrading of collagen type IV. The major protein in the cornea is collagen. Matrix metalloproteinase-9 (MMP-9) is able to degrade collagen type IV from the basement membrane and extracellular matrix (ECM). MMP-9 enzymatic activity is inhibited by the tissue inhibitor of metalloproteinase-1 (TIMP-1). In the present study, we sought to investigate and evaluate the effects of single nucleotide polymorphisms in COL4A3, MMP-9, and TIMP-1 on the risk of KC in an Iranian population sample.
METHODS: This case-control study was performed on 140 KC patients and 150 healthy controls. Genotyping of the COL4A3 rs55703767, MMP-9 rs17576, and TIMP-1 rs6609533 polymorphisms was done using amplification refractory mutation system polymerase chain reaction (ARMS-PCR).
RESULTS: Our findings showed that the rs55703767G/T polymorphism decreased the risk of KC (OR = 0.26, 95% CI = 0.08-0.82, P = 0.022). rs17576A/G, associated with KC and the A allele, was significantly overrepresented in healthy individuals. rs6609533A/G (X-chromosome) increased the risk of KC in females (OR = 2.27, 95% CI = 1.06-4.76, P = 0.036). In males, the allele frequency was not associated with KC risk/protection.
CONCLUSIONS: This study indicates that in our population, the COL4A3 rs55703767 polymorphism decreased the risk of KC. However, the TIMP-1 rs6609533 polymorphism was associated with an increased risk of KC.

Entities:  

Keywords:  COL4A3; Keratoconus; MMP-9; Polymorphism; TIMP-1

Mesh:

Substances:

Year:  2017        PMID: 28197741     DOI: 10.1007/s10384-017-0503-3

Source DB:  PubMed          Journal:  Jpn J Ophthalmol        ISSN: 0021-5155            Impact factor:   2.447


  40 in total

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Authors:  Charles J Malemud
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2.  Analysis of any point mutation in DNA. The amplification refractory mutation system (ARMS).

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5.  Colocalization of the genes for the alpha 3(IV) and alpha 4(IV) chains of type IV collagen to chromosome 2 bands q35-q37.

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6.  Evaluation of possible relationship between COL4A4 gene polymorphisms and risk of keratoconus.

Authors:  Ramin Saravani; Farzaneh Hasanian-Langroudi; Mohammad-Hosein Validad; Davood Yari; Gholamreza Bahari; Mahmood Faramarzi; Mehdi Khateri; Somayeh Bahadoram
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7.  Inhibition of angiogenesis by tissue inhibitor of metalloproteinase.

Authors:  M D Johnson; H R Kim; L Chesler; G Tsao-Wu; N Bouck; P J Polverini
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Review 2.  Association of TIMP-1 and COL4A4 Gene Polymorphisms with Keratoconus in an Iranian Population.

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3.  Collagen cross-linking impact on keratoconus extracellular matrix.

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Review 4.  The Impact of Matrix Metalloproteinase-9 on the Sequential Steps of the Metastatic Process.

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5.  Do age-related macular degeneration genes show association with keratoconus?

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6.  Potential underlying genetic associations between keratoconus and diabetes mellitus.

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7.  Novel Mutations Identified in the Chinese Han Population with Keratoconus by Next-Generation Sequencing.

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