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Literature DB >> 28192751

RETRACTED: Ameliorative potential of linagliptin and/or calcipotriol on bleomycin-induced lung fibrosis: In vivo and in vitro study.

Ahmed M Kabel¹, Maaly A Abd Elmaaboud², Aliaa Atef³, Mohammed H Baali⁴.

Abstract

This article has been retracted: please see Elsevier Policy on Article Withdrawal (https://www.elsevier.com/about/our-business/policies/article-withdrawal). This article has been retracted due to the authors’ plagiarism of text and images from the work of Eman Said Abd-Elkhalek, Hatem Abdel-Rahman Salem, Ghada Mohamed SuddeK, Marwa Ahmed Zaghloul and Ramy Ahmed Abdel-Salam, Faculties of Pharmacy and Medicine, Mansoura University, Mansoura, Egypt.

Entities: Chemical Disease Species

Keywords: Bleomycin; Calcipotriol; Linagliptin; Pulmonary fibrosis; Study

Mesh：

Substances：

Year: 2017 PMID： 28192751 DOI： 10.1016/j.etap.2017.02.008

Source DB: PubMed Journal: Environ Toxicol Pharmacol ISSN： 1382-6689 Impact factor: 4.860

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Cited

2 in total

1. Characterizing drug-related adverse events by joint analysis of biomedical and genomic data: A case study of drug-induced pulmonary fibrosis.

Authors: Alex Jiang; Anil G Jegga
Journal: AMIA Jt Summits Transl Sci Proc Date: 2018-05-18

2. 1α,25-dihydroxyvitamin D3 Attenuates TGF-β-Induced Pro-Fibrotic Effects in Human Lung Epithelial Cells through Inhibition of Epithelial-Mesenchymal Transition.

Authors: Fei Jiang; Yong Yang; Lian Xue; Bingyan Li; Zengli Zhang
Journal: Nutrients Date: 2017-09-06 Impact factor: 5.717

2 in total

RETRACTED: Ameliorative potential of linagliptin and/or calcipotriol on bleomycin-induced lung fibrosis: In vivo and in vitro study.

1. Characterizing drug-related adverse events by joint analysis of biomedical and genomic data: A case study of drug-induced pulmonary fibrosis.

2. 1&#945;,25-dihydroxyvitamin D3 Attenuates TGF-&#946;-Induced Pro-Fibrotic Effects in Human Lung Epithelial Cells through Inhibition of Epithelial-Mesenchymal Transition.

2. 1α,25-dihydroxyvitamin D3 Attenuates TGF-β-Induced Pro-Fibrotic Effects in Human Lung Epithelial Cells through Inhibition of Epithelial-Mesenchymal Transition.