Literature DB >> 28189712

Interleukin-1 receptor antagonist, a biomarker of response to anti-TB treatment in HIV/TB co-infected patients.

Janin Nouhin1, Polidy Pean2, Yoann Madec3, Mathieu F Chevalier4, Celine Didier4, Laurence Borand5, François-Xavier Blanc6, Daniel Scott-Algara4, Didier Laureillard7, Laurence Weiss8.   

Abstract

OBJECTIVES: Despite the high frequency of tuberculosis-associated immune reconstitution inflammatory syndrome (TB-IRIS) in human immunodeficiency virus (HIV)/TB co-infected patients, no diagnostic test is available. Here, we investigated whether monocyte/macrophage activation markers can predict TB-IRIS occurrence and if they are modulated by anti-TB treatment.
METHODS: Frozen plasma was obtained from 127 HIV/TB co-infected adults naïve for antiretroviral therapy, enrolled in the CAMELIA trial, 36 of whom developed TB-IRIS. Concentrations of IL-1Ra, sCD14, and sCD163 were measured at anti-TB treatment onset (baseline), after 8 weeks of anti-TB treatment and at TB-IRIS time.
RESULTS: At baseline, IL-1Ra and sCD14 concentrations were similar in TB-IRIS and non-IRIS patients. sCD163 concentrations, although significantly higher in TB-IRIS patients, did not remain associated with TB-IRIS occurrence in multivariate analysis. At the time of TB-IRIS, patients displayed higher concentrations of IL-1Ra (p = 0.002) and sCD14 (p < 0.001). The most striking result was the significant decrease in IL-1Ra after 8 weeks of anti-TB treatment (median reduction: -63% (p < 0.0001)).
CONCLUSIONS: None of the biomarkers tested was associated with TB-IRIS occurrence. However, repeated measurement of IL-1Ra could help for the diagnosis of TB-IRIS. The substantial reduction of IL-1Ra under treatment suggests that IL-1Ra could be a surrogate biomarker of anti-TB treatment response in HIV-infected patients.
Copyright © 2017 The British Infection Association. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Anti-tuberculosis treatment; Biomarkers; Cambodia; HIV/tuberculosis co-infection; IL-1 receptor antagonist; TB-IRIS

Mesh:

Substances:

Year:  2017        PMID: 28189712     DOI: 10.1016/j.jinf.2017.01.016

Source DB:  PubMed          Journal:  J Infect        ISSN: 0163-4453            Impact factor:   6.072


  3 in total

1.  Changes in inflammatory protein and lipid mediator profiles persist after antitubercular treatment of pulmonary and extrapulmonary tuberculosis: A prospective cohort study.

Authors:  Caian L Vinhaes; Deivide Oliveira-de-Souza; Paulo S Silveira-Mattos; Betania Nogueira; Ruiru Shi; Wang Wei; Xing Yuan; Guolong Zhang; Ying Cai; Clifton E Barry; Laura E Via; Kiyoshi F Fukutani; Bruno B Andrade; Katrin D Mayer-Barber
Journal:  Cytokine       Date:  2019-06-18       Impact factor: 3.861

2.  Lysosomal Cathepsin Release Is Required for NLRP3-Inflammasome Activation by Mycobacterium tuberculosis in Infected Macrophages.

Authors:  Eduardo P Amaral; Nicolas Riteau; Mahtab Moayeri; Nolan Maier; Katrin D Mayer-Barber; Rosana M Pereira; Silvia L Lage; Andre Kubler; William R Bishai; Maria R D'Império-Lima; Alan Sher; Bruno B Andrade
Journal:  Front Immunol       Date:  2018-06-21       Impact factor: 7.561

Review 3.  Advances on the Role and Applications of Interleukin-1 in Tuberculosis.

Authors:  Diogo Silvério; Rute Gonçalves; Rui Appelberg; Margarida Saraiva
Journal:  mBio       Date:  2021-11-23       Impact factor: 7.867

  3 in total

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