Literature DB >> 28179330

Down-regulation of RASA1 Is Associated with Poor Prognosis in Human Hepatocellular Carcinoma.

Yao-Li Chen1,2,3, Wei-Chieh Huang4, Hsin-Lei Yao5, Po-Ming Chen4,6, Ping-Yi Lin2, Fu-Yu Feng7, Pei-Yi Chu8,9,10.   

Abstract

BACKGROUND/AIM: RASA1 (p120RasGAP), encodes Ras GTPase-activating protein 1 and, is a potent tumor suppressor gene that is frequently inactivated in several human cancer types. However, its precise role in hepatocellular carcinoma (HCC) has been blurred.
MATERIALS AND METHODS: We hypothesized that RASA1 plays a crucial role in tumor pathogenesis and progression of HCC. RASA1 expression levels were analyzed in 226 cases of HCC by immunohistochemistry.
RESULTS: It was found that 38.68% (41/106) of the high-grade HCC samples and 54.17% (65/120) of the low-grade HCC samples expressed RASA1 protein. The difference between RASA1 expression in high-grade and low-grade HCC was statistically significant (p=0.02). Additionally, RASA1 high expression was inversely associated with larger tumor size (p<0.001). Although RASA1 is known as a tumor suppressor, its role in overall survival (OS) in HCC is unclear. Kaplan-Meier survival analysis showed that patients with low level of RASA1 expression correlated with a significantly poorer survival compared to those with high level of RASA1 expression.
CONCLUSION: These data support that RASA1 could serve as an independent prognostic marker for HCC patients. Copyright
© 2017, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Entities:  

Keywords:  Liver cancer; RAS-GAP; RASA1; overall survival

Mesh:

Substances:

Year:  2017        PMID: 28179330     DOI: 10.21873/anticanres.11377

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  9 in total

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6.  IFI44L is a novel tumor suppressor in human hepatocellular carcinoma affecting cancer stemness, metastasis, and drug resistance via regulating met/Src signaling pathway.

Authors:  Wei-Chieh Huang; Shiao-Lin Tung; Yao-Li Chen; Po-Ming Chen; Pei-Yi Chu
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  9 in total

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