Literature DB >> 28130758

HLA-C*06:02 Does Not Predispose to Clinical Response Following Long-Term Adalimumab Treatment in Psoriatic Patients: A Retrospective Cohort Study.

Marina Talamonti1, Marco Galluzzo2, Arianna Zangrilli2, Marina Papoutsaki3, Colin Gerard Egan4, Mauro Bavetta2, Sara Tambone5, Maria Concetta Fargnoli5, Luca Bianchi2.   

Abstract

BACKGROUND: The genetic basis of predisposition to psoriasis is recognised; however, the response to psoriasis treatment in patients with different genetic predisposition is poorly understood.
OBJECTIVE: To analyse the presence of the HLA-C*06:02 polymorphism in psoriatic patients treated with adalimumab.
METHODS: Genomic DNA was extracted from whole blood of 122 patients with moderate-to-severe psoriasis treated with adalimumab for 3 years. Genotyping was performed using PCR. Disease severity was assessed by the Psoriasis Area and Severity Index (PASI) at day 0 and after 1, 3, 6, 12, 24 and 36 months. Logistic regression was used to evaluate the association between dependent variables (including HLA-C*06:02 status) and achievement of PASI 50, 75 and 90.
RESULTS: No difference was observed after adalimumab treatment between C*06:02 positive (HLA-C*06:02-POS) patients (n = 46) and C*06:02 negative (HLA-C*06:02-NEG) patients (n = 76) over the 3-year follow-up period in terms of PASI response or time-course when PASI response was achieved. However, a small, but non-statistically significant difference was noted between genotypes for PASI 50 at 1 month (HLA-C*06:02-NEG: 44.7% vs. HLA-C*06:02-POS: 56.5%) and at 3 months (HLA-C*06:02-NEG: 71.1% vs. HLA-C*06:02-POS: 80.4%). Simple logistic regression analysis did not reveal an association between independent variables (including C*06:02 status) and PASI response; however, multivariate regression revealed that gender (females better than males) was associated with achievement of PASI 50 at month 1 (OR 0.34, 95% CI 0.16-0.72, p = 0.005) and of PASI 75 at 3 months (OR 0.36, 95% CI 0.16-0.8, p = 0.012).
CONCLUSION: Adalimumab reduced long-term severity in patients with moderate-severe psoriasis, independent of their HLA-C*06:02 status.

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Year:  2017        PMID: 28130758     DOI: 10.1007/s40291-017-0261-4

Source DB:  PubMed          Journal:  Mol Diagn Ther        ISSN: 1177-1062            Impact factor:   4.074


  33 in total

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Authors:  R B Warren; C E M Griffiths
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Review 2.  Immunopathogenesis of psoriasis.

Authors:  Brian J Nickoloff; Jian-Zhong Qin; Frank O Nestle
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Review 4.  Antidrug antibodies in psoriasis: a systematic review.

Authors:  L Hsu; B T Snodgrass; A W Armstrong
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Review 5.  Pathogenesis and therapy of psoriasis.

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8.  The relationship between tumour necrosis factor (TNF)-α promoter and IL12B/IL-23R genes polymorphisms and the efficacy of anti-TNF-α therapy in psoriasis: a case-control study.

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Review 9.  Sex Differences in the Treatment of Psoriatic Arthritis: A Systematic Literature Review.

Authors:  Elena Generali; Carlo A Sciré; Luca Cantarini; Carlo Selmi
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10.  IL12B (p40) Gene Polymorphisms Contribute to Ustekinumab Response Prediction in Psoriasis.

Authors:  Marco Galluzzo; Andreea Nicoleta Boca; Elisabetta Botti; Concetta Potenza; Giovanna Malara; Piergiorgio Malagoli; Stefan Vesa; Sergio Chimenti; Anca Dana Buzoianu; Marina Talamonti; Antonio Costanzo
Journal:  Dermatology       Date:  2015-12-18       Impact factor: 5.366

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Journal:  Mol Diagn Ther       Date:  2019-10       Impact factor: 4.074

2.  HLA-C*06:02 allele can influence clinical efficacy of certolizumab pegol?

Authors:  Annunziata Dattola; Elisabetta Botti; Marco Galluzzo; Dante Raffaele Caro Caposiena; Sara Mazzilli; Elena Campione; Gennaro Melino; Eleonora Candi; Luca Bianchi
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Review 3.  Influence of Genetic Polymorphisms on Response to Biologics in Moderate-to-Severe Psoriasis.

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