Literature DB >> 28124644

Differences in innate immune response gene regulation in the middle ear of children who are otitis prone and in those not otitis prone.

Ravinder Kaur1, Janet Casey, Michael Pichichero.   

Abstract

OBJECTIVE: Acute otitis media (AOM) causes an inflammatory response in the middle ear. We assessed differences in innate immune responses involved in bacterial defense at onset of AOM in children who were stringently defined as otitis prone (sOP) and children not otitis prone (NOP). STUDY
DESIGN: Innate immune genes analysis from middle ear fluid (MEF) samples of children.
METHODS: Genes of toll-like receptors (TLR), nod-like and retinoic acid-inducible gene-I-like receptors, downstream effectors important for inflammation and apoptosis, including cytokines and chemokines, were studied from MEF samples by using a real-time polymerase chain reaction array. Protein levels of differentially regulated genes were measured by Luminex.
RESULTS: Gene expression in MEF among children who were sOP was significantly different in upregulation of interleukin 8, secretory leukocyte peptidase inhibitor, and chemokine (C-C motif) ligand 3, and in downregulation of interferon regulatory factor 7 and its related signaling molecules interferon alpha, Toll-like receptor adaptor molecule 2, chemokine (C-C motif) ligand 5, and mitogen-activated protein kinase 8 compared with children who were NOP. Differences in innate gene regulation were similar when AOM was caused by Streptococcus pneumoniae or nontypeable Haemophilus influenzae.
CONCLUSION: Innate-immune response genes are differentially regulated in children who were sOP compared with children with NOP.

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Year:  2016        PMID: 28124644      PMCID: PMC5108842          DOI: 10.2500/ajra.2016.30.4393

Source DB:  PubMed          Journal:  Am J Rhinol Allergy        ISSN: 1945-8932            Impact factor:   2.467


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