Literature DB >> 28100690

Oxytocin Treatment May Improve Infant Feeding and Social Skills in Prader-Willi Syndrome.

Nancie J MacIver1.   

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Year:  2017        PMID： 28100690      PMCID： PMC5260161          DOI： 10.1542/peds.2016-3833

Source DB:  PubMed          Journal:  Pediatrics        ISSN： 0031-4005            Impact factor:   7.124


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  8 in total

1.  A single postnatal injection of oxytocin rescues the lethal feeding behaviour in mouse newborns deficient for the imprinted Magel2 gene.

Authors:  Fabienne Schaller; Françoise Watrin; Rachel Sturny; Annick Massacrier; Pierre Szepetowski; Françoise Muscatelli
Journal:  Hum Mol Genet       Date:  2010-09-28       Impact factor: 6.150

2.  A double-blind randomized controlled trial of oxytocin nasal spray in Prader Willi syndrome.

Authors:  Stewart L Einfeld; Ellie Smith; Iain S McGregor; Kate Steinbeck; John Taffe; Lauren J Rice; Siân K Horstead; Naomi Rogers; M Antoinette Hodge; Adam J Guastella
Journal:  Am J Med Genet A       Date:  2014-06-30       Impact factor: 2.802

3.  Oxytocin may be useful to increase trust in others and decrease disruptive behaviours in patients with Prader-Willi syndrome: a randomised placebo-controlled trial in 24 patients.

Authors:  Maïthe Tauber; Carine Mantoulan; Pierre Copet; Joseba Jauregui; Genevieve Demeer; Gwenaëlle Diene; Bernadette Rogé; Virginie Laurier; Virginie Ehlinger; Catherine Arnaud; Catherine Molinas; Denise Thuilleaux
Journal:  Orphanet J Rare Dis       Date:  2011-06-24       Impact factor: 4.123

4.  Promising effects of oxytocin on social and food-related behaviour in young children with Prader-Willi syndrome: a randomized, double-blind, controlled crossover trial.

Authors:  R J Kuppens; S H Donze; A C S Hokken-Koelega
Journal:  Clin Endocrinol (Oxf)       Date:  2016-08-26       Impact factor: 3.478

5.  An Early Postnatal Oxytocin Treatment Prevents Social and Learning Deficits in Adult Mice Deficient for Magel2, a Gene Involved in Prader-Willi Syndrome and Autism.

Authors:  Hamid Meziane; Fabienne Schaller; Sylvian Bauer; Claude Villard; Valery Matarazzo; Fabrice Riet; Gilles Guillon; Daniel Lafitte; Michel G Desarmenien; Maithé Tauber; Françoise Muscatelli
Journal:  Biol Psychiatry       Date:  2014-11-20       Impact factor: 13.382

6.  Alterations in the hypothalamic paraventricular nucleus and its oxytocin neurons (putative satiety cells) in Prader-Willi syndrome: a study of five cases.

Authors:  D F Swaab; J S Purba; M A Hofman
Journal:  J Clin Endocrinol Metab       Date:  1995-02       Impact factor: 5.958

Review 7.  Prader Willi Syndrome: Genetics, Metabolomics, Hormonal Function, and New Approaches to Therapy.

Authors:  Krystal A Irizarry; Mark Miller; Michael Freemark; Andrea M Haqq
Journal:  Adv Pediatr       Date:  2016-08

8.  Truncating mutations of MAGEL2 cause Prader-Willi phenotypes and autism.

Authors:  Christian P Schaaf; Manuel L Gonzalez-Garay; Fan Xia; Lorraine Potocki; Karen W Gripp; Baili Zhang; Brock A Peters; Mark A McElwain; Radoje Drmanac; Arthur L Beaudet; C Thomas Caskey; Yaping Yang
Journal:  Nat Genet       Date:  2013-09-29       Impact factor: 38.330
  8 in total

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