Literature DB >> 28097385

Two Phase 1 dose-escalation studies exploring multiple regimens of litronesib (LY2523355), an Eg5 inhibitor, in patients with advanced cancer.

Jeffrey R Infante1,2, Amita Patnaik3, Claire F Verschraegen4,5, Anthony J Olszanski6, Montaser Shaheen4, Howard A Burris7,8, Anthony W Tolcher3, Kyriakos P Papadopoulos3, Muralidhar Beeram3, Scott M Hynes9, Jennifer Leohr9, Aimee Bence Lin9, Lily Q Li9, Anna McGlothlin9,10, Daphne L Farrington9, Eric H Westin9,11, Roger B Cohen6,12.   

Abstract

PURPOSE: This first-in-human report examined the recommended Phase 2 dose and schedule of litronesib, a selective allosteric kinesin Eg5 inhibitor.
METHODS: Two concurrent dose-escalation studies investigated litronesib across the dose range of 0.125-16 mg/m2/day, evaluating the following schedules of administration on a 21-day cycle: Days 1, 2, 3; Days 1, 5, 9; Days 1, 8; Days 1, 5; or Days 1, 4, with or without pegfilgrastim. Best overall response was defined per Response Evaluation Criteria in Solid Tumors (RECIST Version 1.0). Pharmacokinetic (PK) evaluations were performed. Exploratory PK/pharmacodynamic analyses investigated the relationship between litronesib plasma exposure and changes in phosphohistone H3 (pHH3) levels.
RESULTS: One hundred and seventeen patients with advanced malignancies were enrolled. Neutropenia was the primary dose-limiting toxicity. Prophylactic pegfilgrastim reduced neutropenia frequency and severity, allowing administration of higher litronesib doses, but increases in the incidences of mucositis and stomatitis were observed. Among 86 response-evaluable patients, 2 patients (2%) achieved partial response, both on the Days 1, 2, 3 regimen (5 and 6 mg/m2/day with pegfilgrastim), and 17 patients (20%) maintained stable disease for ≥6 cycles. Dose-dependent increases in litronesib plasma exposure were observed, with minor intra- and inter-cycle accumulation, along with exposure-dependent increases in pHH3 expression in tumor and skin biopsies.
CONCLUSIONS: On the basis of the results of these studies, two regimens were selected for Phase 2 exploration: 6 mg/m2/day on Days 1, 2, 3 plus pegfilgrastim and 8 mg/m2/day on Days 1, 5, 9 plus pegfilgrastim, both on a 21-day cycle.

Entities:  

Keywords:  Antimitotic; Eg5; KSP inhibitor; Kinesin spindle protein (KSP); Phase 1

Mesh:

Substances:

Year:  2017        PMID: 28097385     DOI: 10.1007/s00280-016-3205-5

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  7 in total

Review 1.  A New Way to Treat Brain Tumors: Targeting Proteins Coded by Microcephaly Genes?: Brain tumors and microcephaly arise from opposing derangements regulating progenitor growth. Drivers of microcephaly could be attractive brain tumor targets.

Authors:  Patrick Y Lang; Timothy R Gershon
Journal:  Bioessays       Date:  2018-03-26       Impact factor: 4.345

2.  Exploratory comparisons between different anti-mitotics in clinically-used drug combination in triple negative breast cancer.

Authors:  Bruna Cândido Guido; Douglas Cardoso Brandão; Ana Luisa Augusto Barbosa; Monique Jacob Xavier Vianna; Lucas Faro; Luciana Machado Ramos; Fabíola Nihi; Márcio Botelho de Castro; Brenno A D Neto; José Raimundo Corrêa; Sônia Nair Báo
Journal:  Oncotarget       Date:  2021-09-14

3.  Overexpression of kinesin superfamily members as prognostic biomarkers of breast cancer.

Authors:  Tian-Fu Li; Hui-Juan Zeng; Zhen Shan; Run-Yi Ye; Tuck-Yun Cheang; Yun-Jian Zhang; Si-Hong Lu; Qi Zhang; Nan Shao; Ying Lin
Journal:  Cancer Cell Int       Date:  2020-04-15       Impact factor: 5.722

Review 4.  Thiadiazole derivatives as anticancer agents.

Authors:  Monika Szeliga
Journal:  Pharmacol Rep       Date:  2020-09-03       Impact factor: 3.024

Review 5.  Kinesin spindle protein inhibitors in cancer: from high throughput screening to novel therapeutic strategies.

Authors:  Rand Shahin; Salah Aljamal
Journal:  Future Sci OA       Date:  2022-02-21

6.  Death receptor 6 promotes ovarian cancer cell migration through KIF11.

Authors:  Bianhua Shi; Jiayu Bao; Yongbin Liu; Juan Shi
Journal:  FEBS Open Bio       Date:  2018-08-07       Impact factor: 2.693

7.  Suppression of KIF22 Inhibits Cell Proliferation and Xenograft Tumor Growth in Tongue Squamous Cell Carcinoma.

Authors:  Yi Liu; Rong-Hua Li; Gang Ren; Jin Jiang
Journal:  Biomed Res Int       Date:  2020-01-21       Impact factor: 3.411

  7 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.