Literature DB >> 27994602

Vasodilation and Exercise Capacity in Patients with End-Stage Renal Disease: A Prospective Proof-of-Concept Study.

Dirk Habedank1, Joerg C Schefold2, Carolin Bernhardt3, Tim Karhausen3, Wolfram Doehner4, Stefan D Anker5, Petra Reinke6.   

Abstract

BACKGROUND: Previous data have pointed to the fact that vascular function is significantly impaired in patients with end-stage renal disease (ESRD). We aimed to better characterise vasodilation and exercise capacity in both ESRD and chronic heart failure (CHF) patients.
METHODS: A total of 30 ESRD patients (23 male; mean age 45.7 ± 9.9 years) were included in a prospective proof-of-concept study at a tertiary care academic centre. The patients underwent forearm venous plethysmography with post-ischaemic peak blood flow (PF) and flow-dependent flow (FDF) testing as well as cardiopulmonary exercise testing during the morning of the day following the last haemodialysis. After matching for age, gender, and body mass index, the data were compared to 30 patients with CHF and 20 age-matched healthy controls.
RESULTS: PF in ESRD patients was reduced when compared to that in CHF patients (12.5 ± 4.2 vs. 15.6 ± 6.9 ml/100 ml/min; p = 0.048) and healthy controls (26.4 ± 9.3 ml/100 ml/min; p < 0.001). When compared to controls, FDF was significantly reduced in ESRD patients (7.6 ± 3.1 vs. 6.0 ± 2.5 ml/100 ml/min; p = 0.03), but not in CHF patients, whereas resting blood flow did not differ between the ESRD, CHF, and healthy control groups. In contrast to indices of vasodilative capacity, maximum exercise capacity (peakVO2) was higher in ESRD when compared to CHF patients (23.8 ± 7.3 vs. 18.8 ± 5.2 ml/min/kg), but significantly impaired when compared to controls (32.8 ± 6.7 ml/min/kg; p < 0.001).
CONCLUSION: In this proof-of-concept study, exercise capacity was relatively preserved, while vasodilative capacity was substantially impaired in ESRD patients. Additional studies are warranted to examine the underlying mechanisms and potential clinical implications of our findings.

Entities:  

Keywords:  Chronic heart failure; Chronic kidney disease; Exercise; Inflammation; Sepsis; Vasodilation

Year:  2016        PMID: 27994602      PMCID: PMC5159734          DOI: 10.1159/000449174

Source DB:  PubMed          Journal:  Cardiorenal Med        ISSN: 1664-5502            Impact factor:   2.041


  33 in total

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8.  Validation of peak exercise oxygen consumption and the Heart Failure Survival Score for serial risk stratification in advanced heart failure.

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9.  Exercise capacity as a predictor of survival among ambulatory patients with end-stage renal disease.

Authors:  Kathy E Sietsema; Antonino Amato; Sharon G Adler; Eric P Brass
Journal:  Kidney Int       Date:  2004-02       Impact factor: 10.612

10.  Increased indoleamine 2,3-dioxygenase (IDO) activity and elevated serum levels of tryptophan catabolites in patients with chronic kidney disease: a possible link between chronic inflammation and uraemic symptoms.

Authors:  Jörg C Schefold; Jan-Philip Zeden; Christina Fotopoulou; Stephan von Haehling; Rene Pschowski; Dietrich Hasper; Hans-Dieter Volk; Christine Schuett; Petra Reinke
Journal:  Nephrol Dial Transplant       Date:  2009-01-20       Impact factor: 5.992

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Review 1.  The impact of exercise on physical function, cardiovascular outcomes and quality of life in chronic kidney disease patients: a systematic review.

Authors:  Baris Afsar; Dimitrie Siriopol; Gamze Aslan; Ozgur C Eren; Tuncay Dagel; Ugur Kilic; Asiye Kanbay; Alexandru Burlacu; Adrian Covic; Mehmet Kanbay
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