Literature DB >> 27975209

Critical role for GLP-1 in symptomatic post-bariatric hypoglycaemia.

Colleen M Craig1, Li-Fen Liu2, Carolyn F Deacon3, Jens J Holst3, Tracey L McLaughlin2.   

Abstract

AIMS/HYPOTHESIS: Post-bariatric hypoglycaemia (PBH) is a rare, but severe, metabolic disorder arising months to years after bariatric surgery. It is characterised by symptomatic postprandial hypoglycaemia, with inappropriately elevated insulin concentrations. The relative contribution of exaggerated incretin hormone signalling to dysregulated insulin secretion and symptomatic hypoglycaemia is a subject of ongoing inquiry. This study was designed to test the hypothesis that PBH and associated symptoms are primarily mediated by glucagon-like peptide-1 (GLP-1).
METHODS: We conducted a double-blinded crossover study wherein eight participants with confirmed PBH were assigned in random order to intravenous infusion of the GLP-1 receptor (GLP-1r) antagonist. Exendin (9-39) (Ex-9), or placebo during an OGTT on two separate days at the Stanford University Clinical and Translational Research Unit. Metabolic, symptomatic and pharmacokinetic variables were evaluated. Results were compared with a cohort of BMI- and glucose-matched non-surgical controls (NSCs).
RESULTS: Infusion of Ex-9 decreased the time to peak glucose and rate of glucose decline during OGTT, and raised the postprandial nadir by over 70%, normalising it relative to NSCs and preventing hypoglycaemia in all PBH participants. Insulin AUC and secretion rate decreased by 57% and 71% respectively, and peak postprandial insulin was normalised relative to NSCs. Autonomic and neuroglycopenic symptoms were significantly reduced during Ex-9 infusion. CONCLUSIONS/
INTERPRETATION: GLP-1r blockade prevented hypoglycaemia in 100% of individuals, normalised beta cell function and reversed neuroglycopenic symptoms, supporting the conclusion that GLP-1 plays a primary role in mediating hyperinsulinaemic hypoglycaemia in PBH. Competitive antagonism at the GLP-1r merits consideration as a therapeutic strategy. TRIAL REGISTRATION: ClinicalTrials.gov NCT02550145.

Entities:  

Keywords:  Bariatric complication; GLP-1; Hyperinsulinaemic hypoglycaemia; Hypoglycaemia; Late dumping syndrome; NIPHS; Nesidioblastosis; Neuroglycopenia; Noninsulinoma pancreatogenous hypoglycaemia syndrome; Post-bariatric hypoglycaemia; RYGB; Roux-en-Y gastric bypass

Mesh:

Substances:

Year:  2016        PMID: 27975209      PMCID: PMC5300915          DOI: 10.1007/s00125-016-4179-x

Source DB:  PubMed          Journal:  Diabetologia        ISSN: 0012-186X            Impact factor:   10.122


  47 in total

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8.  Postprandial hypoglycemia after gastric bypass surgery: from pathogenesis to diagnosis and treatment.

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9.  Insights from the Impact of Meal Composition on Glucose Profile Towards Post-bariatric Hypoglycemia Management.

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10.  A Randomized, Placebo-Controlled Double-Blind Trial of a Closed-Loop Glucagon System for Postbariatric Hypoglycemia.

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