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Literature DB >> 27942530

Drugs targeting dynamin can restore cytoskeleton and focal contact alterations of urinary podocytes derived from patients with nephrotic syndrome.

Janina Müller-Deile¹, Beina Teng¹, Heiko Schenk¹, Hermann Haller¹, Jochen Reiser², Sanja Sever³, Mario Schiffer¹.

Abstract

Entities: Disease Species

Year: 2016 PMID： 27942530 PMCID： PMC5124626 DOI： 10.21037/atm.2016.10.72

Source DB: PubMed Journal: Ann Transl Med ISSN： 2305-5839

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24 in total

1. Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis.

Authors: J M Kaplan; S H Kim; K N North; H Rennke; L A Correia; H Q Tong; B J Mathis; J C Rodríguez-Pérez; P G Allen; A H Beggs; M R Pollak
Journal: Nat Genet Date: 2000-03 Impact factor: 38.330

2. Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney disease.

Authors: Sanja Sever; Mehmet M Altintas; Sharif R Nankoe; Clemens C Möller; David Ko; Changli Wei; Joel Henderson; Elizabetta C del Re; Lianne Hsing; Ann Erickson; Clemens D Cohen; Matthias Kretzler; Dontscho Kerjaschki; Alexander Rudensky; Boris Nikolic; Jochen Reiser
Journal: J Clin Invest Date: 2007-08 Impact factor: 14.808

3. MYO1E mutations and childhood familial focal segmental glomerulosclerosis.

Authors: Caterina Mele; Paraskevas Iatropoulos; Roberta Donadelli; Andrea Calabria; Ramona Maranta; Paola Cassis; Simona Buelli; Susanna Tomasoni; Rossella Piras; Mira Krendel; Serena Bettoni; Marina Morigi; Massimo Delledonne; Carmine Pecoraro; Isabella Abbate; Maria Rosaria Capobianchi; Friedhelm Hildebrandt; Edgar Otto; Franz Schaefer; Fabio Macciardi; Fatih Ozaltin; Sevinc Emre; Tulin Ibsirlioglu; Ariela Benigni; Giuseppe Remuzzi; Marina Noris
Journal: N Engl J Med Date: 2011-07-14 Impact factor: 91.245

4. Urinary podocytes in primary focal segmental glomerulosclerosis.

Authors: M Hara; T Yanagihara; I Kihara
Journal: Nephron Date: 2001-11 Impact factor: 2.847

5. Pathogenesis of polycation-induced alterations ("fusion") of glomerular epithelium.

Authors: M W Seiler; H G Rennke; M A Venkatachalam; R S Cotran
Journal: Lab Invest Date: 1977-01 Impact factor: 5.662

Review 6. Integrin-linked kinase in renal disease: connecting cell-matrix interaction to the cytoskeleton.

Authors: Simone Monika Blattner; Matthias Kretzler
Journal: Curr Opin Nephrol Hypertens Date: 2005-07 Impact factor: 2.894

7. Urine podocyte mRNAs mark progression of renal disease.

Authors: Yuji Sato; Bryan L Wharram; Sang Koo Lee; Larysa Wickman; Meera Goyal; Madhusudan Venkatareddy; Jai Won Chang; Jocelyn E Wiggins; Chrysta Lienczewski; Matthias Kretzler; Roger C Wiggins
Journal: J Am Soc Nephrol Date: 2009-04-23 Impact factor: 10.121

8. Role of dynamin, synaptojanin, and endophilin in podocyte foot processes.

Authors: Keita Soda; Daniel M Balkin; Shawn M Ferguson; Summer Paradise; Ira Milosevic; Silvia Giovedi; Laura Volpicelli-Daley; Xuefei Tian; Yumei Wu; Hong Ma; Sung Hyun Son; Rena Zheng; Gilbert Moeckel; Ottavio Cremona; Lawrence B Holzman; Pietro De Camilli; Shuta Ishibe
Journal: J Clin Invest Date: 2012-11-26 Impact factor: 14.808

Drugs targeting dynamin can restore cytoskeleton and focal contact alterations of urinary podocytes derived from patients with nephrotic syndrome.

1. Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis.

2. Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney disease.

3. MYO1E mutations and childhood familial focal segmental glomerulosclerosis.

4. Urinary podocytes in primary focal segmental glomerulosclerosis.

5. Pathogenesis of polycation-induced alterations ("fusion") of glomerular epithelium.

Review 6. Integrin-linked kinase in renal disease: connecting cell-matrix interaction to the cytoskeleton.

7. Urine podocyte mRNAs mark progression of renal disease.

8. Role of dynamin, synaptojanin, and endophilin in podocyte foot processes.

Review 9. Podocyte biology and pathogenesis of kidney disease.

10. Mutations in the formin gene INF2 cause focal segmental glomerulosclerosis.

Review 1. Podocytes from the diagnostic and therapeutic point of view.

2. Mutation of microphthalmia-associated transcription factor (mitf) in zebrafish sensitizes for glomerulopathy.