Fang Yu1, Andrew Steven2, Lee Birnbaum3, Wilson Altmeyer4. 1. Department of Radiology, Massachusetts General Hospital, 55, Fruit Street, Boston, MA 02114, USA. Electronic address: frankfy21@gmail.com. 2. Department of Radiology, University of Maryland Medical System, Baltimore, Maryland, USA. 3. Department of Neurology and Neurosurgery, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA. 4. Department of Radiology, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
Abstract
INTRODUCTION: Hyperglycemia can induce hemichorea-hemiballism, especially in elderly type II diabetics. CT and MRI findings include hyperdensity and T1-shortening in the contralateral lentiform nucleus, respectively. This study explores the associated imaging findings on T2*-based sequences. METHODS: Six patients with clinically documented hyperglycemia-induced hemichorea-hemiballism who had undergone MR imaging with a T2*-based sequence (T2* gradient echo or susceptibility-weighted imaging) were included in this retrospective case series. RESULTS: All six patients demonstrated T1-shortening contralateral to their hemichorea-hemiballism. T2*-based sequences demonstrated unilateral hypointense signal within the striatum in four patients. One patient had mild bilateral striatal hyperintensities, while another did not show significant signal changes. CONCLUSION: It is important for the radiologist to be aware of the signal changes that can be seen on T2*-based sequences in hyperglycemia-induced hemochorea-hemiballism.
INTRODUCTION:Hyperglycemia can induce hemichorea-hemiballism, especially in elderly type II diabetics. CT and MRI findings include hyperdensity and T1-shortening in the contralateral lentiform nucleus, respectively. This study explores the associated imaging findings on T2*-based sequences. METHODS: Six patients with clinically documented hyperglycemia-induced hemichorea-hemiballism who had undergone MR imaging with a T2*-based sequence (T2* gradient echo or susceptibility-weighted imaging) were included in this retrospective case series. RESULTS: All six patients demonstrated T1-shortening contralateral to their hemichorea-hemiballism. T2*-based sequences demonstrated unilateral hypointense signal within the striatum in four patients. One patient had mild bilateral striatal hyperintensities, while another did not show significant signal changes. CONCLUSION: It is important for the radiologist to be aware of the signal changes that can be seen on T2*-based sequences in hyperglycemia-induced hemochorea-hemiballism.