Literature DB >> 27804235

A phase IIa study of afuresertib, an oral pan-AKT inhibitor, in patients with Langerhans cell histiocytosis.

Robert J Arceci1, Carl E Allen2, Ira J Dunkel3, Eric Jacobsen4, James Whitlock5, Robert Vassallo6, Shannon R Morris7,8, Alison Portnoy9,10, Beth Ann Reedy9, Deborah A Smith7,11, Robert Noble9, Amy Murnane9, Mark Cornfeld12,13, Carlos Rodriguez-Galindo4, Mark L Heaney3,14, Kenneth McClain2, Sarah Vaiselbuh15.   

Abstract

BACKGROUND: Langerhans cell histiocytosis (LCH) is a clonal neoplasm characterized by widely varied clinical presentations, including multisystem involvement and systemic inflammatory symptoms. The AKT pathway is relevant to survival and proliferation of dendritic cells, and is also often upregulated in hematopoietic malignancies. A clinical response in an adult patient with LCH participating in the first-in-human trial of afuresertib prompted this prospective trial. PROCEDURE: The population in the current study included treatment-naïve (n = 7) and recurrent/refractory patients with LCH (n = 10), who received oral afuresertib (125 mg). The majority of patients were treated for > 24 weeks, with four patients receiving treatment for > 48 weeks.
RESULTS: Pharmacokinetic analysis showed similar exposures in previously reported patients with other hematologic malignancies. Primary drug-related toxicities included Grade 1/2 nausea, diarrhea, dyspepsia, and vomiting. Grade 3 toxicities included fatigue, diarrhea, and pain (one of each). Another severe adverse event involved soft tissue necrosis. The overall response rate in evaluable subjects was 33% in treatment-naïve patients and 28% in patients with recurrent/refractory disease, which did not meet the predefined Bayesian criteria for efficacy.
CONCLUSION: Afuresertib has clinical activity in some patients with newly diagnosed and advanced LCH.
© 2016 Wiley Periodicals, Inc.

Entities:  

Keywords:  AKT inhibitor; Langerhans cell histiocytosis; afuresertib

Mesh:

Substances:

Year:  2016        PMID: 27804235     DOI: 10.1002/pbc.26325

Source DB:  PubMed          Journal:  Pediatr Blood Cancer        ISSN: 1545-5009            Impact factor:   3.167


  5 in total

1.  Mutually exclusive recurrent somatic mutations in MAP2K1 and BRAF support a central role for ERK activation in LCH pathogenesis.

Authors:  Rikhia Chakraborty; Oliver A Hampton; Xiaoyun Shen; Stephen J Simko; Albert Shih; Harshal Abhyankar; Karen Phaik Har Lim; Kyle R Covington; Lisa Trevino; Ninad Dewal; Donna M Muzny; Harshavardhan Doddapaneni; Jianhong Hu; Linghua Wang; Philip J Lupo; M John Hicks; Diana L Bonilla; Karen C Dwyer; Marie-Luise Berres; Poulikos I Poulikakos; Miriam Merad; Kenneth L McClain; David A Wheeler; Carl E Allen; D Williams Parsons
Journal:  Blood       Date:  2014-09-08       Impact factor: 22.113

2.  KRASG12D expression in lung-resident myeloid cells promotes pulmonary LCH-like neoplasm sensitive to statin treatment.

Authors:  Tamihiro Kamata; Susan Giblett; Catrin Pritchard
Journal:  Blood       Date:  2017-05-26       Impact factor: 22.113

Review 3.  Molecular target: pan-AKT in gastric cancer.

Authors:  Byung Woog Kang; Ian Chau
Journal:  ESMO Open       Date:  2020-09

Review 4.  The Akt pathway in oncology therapy and beyond (Review).

Authors:  George Mihai Nitulescu; Maryna Van De Venter; Georgiana Nitulescu; Anca Ungurianu; Petras Juzenas; Qian Peng; Octavian Tudorel Olaru; Daniela Grădinaru; Aristides Tsatsakis; Dimitris Tsoukalas; Demetrios A Spandidos; Denisa Margina
Journal:  Int J Oncol       Date:  2018-10-16       Impact factor: 5.650

5.  BRAF V600E and Pten deletion in mice produces a histiocytic disorder with features of Langerhans cell histiocytosis.

Authors:  David S Nelson; Ryan L Marano; Yechaan Joo; Sara Y Tian; Bhumi Patel; Daniel H Kaplan; Mark J Shlomchik; Kristen Stevenson; Roderick T Bronson; Barrett J Rollins
Journal:  PLoS One       Date:  2019-09-17       Impact factor: 3.240

  5 in total

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