Literature DB >> 27798053

Relationship between aetiology and left ventricular systolic dysfunction in hypertrophic cardiomyopathy.

Stefania Rosmini1,2, Elena Biagini2, Costantinos O'Mahony1,3, Heerajnarain Bulluck1,3, Niccolo' Ruozi2, Luis R Lopes3,4,5, Oliver Guttmann1,3, Patricia Reant6, Cristina C Quarta2,7, Antonis Pantazis3, Maria Tome-Esteban3, William J Mckenna3, Claudio Rapezzi2, Perry M Elliott1,3.   

Abstract

BACKGROUND: Severe left ventricular (LV) systolic dysfunction is an uncommon complication of hypertrophic cardiomyopathy (HCM) that is associated with poor prognosis. Small observational series suggest that patients with rare causes of HCM are more likely to develop systolic impairment than those with idiopathic disease or mutations in cardiac sarcomeric protein genes. The aim of this study was to test this hypothesis by comparing the prevalence of systolic dysfunction and its impact on prognosis in patients with different causes of HCM. METHODS AND
RESULTS: 1697 patients (52 (40-63) years, 1160 (68%) males) with HCM followed at two European referral centres were studied. Diagnosis of specific aetiologies was made on the basis of clinical examination, cardiac imaging and targeted genetic and biochemical testing. The primary survival outcome was all-cause mortality or heart transplantation (HTx) for end-stage heart failure (HF). Secondary outcomes were HF-related death, sudden cardiac death, stroke-related death and non-cardiovascular death. Systolic dysfunction (LV ejection fraction <50% by two-dimensional (2D) echocardiography) at first evaluation was more frequent in rare phenocopies than in idiopathic or sarcomeric HCM (105/409 (26%) vs 40/1288 (3%), respectively (p<0.0001)). All-cause death/HTx and HF-related death were more frequent in rare phenocopies compared with idiopathic or sarcomeric HCM (p<0.0001). All-cause mortality and HF-related death were highest in patients with cardiac amyloidosis (p<0.0001).
CONCLUSIONS: In adults with HCM, LV systolic dysfunction is more frequent in those with rare phenocopies. When combined with age at presentation, it is a marker for specific aetiologies and is associated with poorer long-term survival. Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/.

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Year:  2016        PMID: 27798053     DOI: 10.1136/heartjnl-2016-310138

Source DB:  PubMed          Journal:  Heart        ISSN: 1355-6037            Impact factor:   5.994


  8 in total

Review 1.  Precision medicine for cardiovascular disease : Learning lessons from cardiomyopathies.

Authors:  F Sedaghat-Hamedani; H A Katus; B Meder
Journal:  Herz       Date:  2018-03       Impact factor: 1.443

Review 2.  Beyond Sarcomeric Hypertrophic Cardiomyopathy: How to Diagnose and Manage Phenocopies.

Authors:  Maurizio Pieroni; Michele Ciabatti; Elisa Saletti; Valentina Tavanti; Pasquale Santangeli; Lucia Martinese; Francesco Liistro; Iacopo Olivotto; Leonardo Bolognese
Journal:  Curr Cardiol Rep       Date:  2022-09-02       Impact factor: 3.955

Review 3.  Fabry Disease and the Heart: A Comprehensive Review.

Authors:  Olga Azevedo; Filipa Cordeiro; Miguel Fernandes Gago; Gabriel Miltenberger-Miltenyi; Catarina Ferreira; Nuno Sousa; Damião Cunha
Journal:  Int J Mol Sci       Date:  2021-04-23       Impact factor: 5.923

4.  Mutations in TRIM63 cause an autosomal-recessive form of hypertrophic cardiomyopathy.

Authors:  Joel Salazar-Mendiguchía; Juan Pablo Ochoa; Julian Palomino-Doza; Fernando Domínguez; Carles Díez-López; Mohammed Akhtar; Soraya Ramiro-León; María M Clemente; Antonia Pérez-Cejas; María Robledo; Iria Gómez-Díaz; María Luisa Peña-Peña; Vicente Climent; Francisco Salmerón-Martínez; Celestino Hernández; Pablo E García-Granja; M Victoria Mogollón; Ivonne Cárdenas-Reyes; Marcos Cicerchia; Diego García-Giustiniani; Arsonval Lamounier; Belén Gil-Fournier; Felícitas Díaz-Flores; Rafael Salguero; Luis Santomé; Petros Syrris; Montse Olivé; Pablo García-Pavía; Martín Ortiz-Genga; Perry M Elliott; Lorenzo Monserrat
Journal:  Heart       Date:  2020-05-25       Impact factor: 5.994

5.  Heterozygous junctophilin-2 (JPH2) p.(Thr161Lys) is a monogenic cause for HCM with heart failure.

Authors:  Sari U M Vanninen; Krista Leivo; Eija H Seppälä; Katriina Aalto-Setälä; Olli Pitkänen; Piia Suursalmi; Antti-Pekka Annala; Ismo Anttila; Tero-Pekka Alastalo; Samuel Myllykangas; Tiina M Heliö; Juha W Koskenvuo
Journal:  PLoS One       Date:  2018-09-20       Impact factor: 3.240

Review 6.  The genetics of hypertrophic cardiomyopathy.

Authors:  Mohammed Akhtar; Perry Elliott
Journal:  Glob Cardiol Sci Pract       Date:  2018-08-12

Review 7.  Anderson-Fabry disease in heart failure.

Authors:  M M Akhtar; P M Elliott
Journal:  Biophys Rev       Date:  2018-06-16

8.  Does left ventricular function predict cardiac outcome in Anderson-Fabry disease?

Authors:  Letizia Spinelli; Giuseppe Giugliano; Antonio Pisani; Massimo Imbriaco; Eleonora Riccio; Camilla Russo; Alberto Cuocolo; Bruno Trimarco; Giovanni Esposito
Journal:  Int J Cardiovasc Imaging       Date:  2020-11-19       Impact factor: 2.357

  8 in total

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