Monish M Maharaj1, Kevin Phan2, Soumya Hariswamy3, Prashanth J Rao4. 1. NeuroSpine Surgery Research Group (NSURG), Sydney, Australia; ; Faculty of Medicine, University of New South Wales, Sydney, Australia. 2. NeuroSpine Surgery Research Group (NSURG), Sydney, Australia; ; Faculty of Medicine, University of Sydney, Sydney, Australia. 3. Faculty of Medicine, University of Sydney, Sydney, Australia. 4. NeuroSpine Surgery Research Group (NSURG), Sydney, Australia; ; Department of Neurosurgery, Prince of Wales Hospital, Sydney, Australia.
Abstract
BACKGROUND: Surfers myelopathy can be a rapidly devastating disease and little is known surrounding the pathophysiology of the condition. Although the classical pattern of illness has been well reported, it has never been observed in a non-surfing setting. METHODS: A 51-year-old demolition worker presented with acute non-traumatic myelopathy. Clinical examination revealed sensory loss to the level of L2. T2-MRI and MRI-DWI revealed a hyperintense signal suggestive of an ischaemic event. A diagnosis of surfer's myelopathy was made and he was commenced on steroid therapy. RESULTS: Following steroid therapy and fluid management the patient was discharged after 6 days with minor anaethesia but significant overall neurological improvement. CONCLUSIONS: Diagnosis of SM requires a thorough history, clinical examination and imaging (MRI, MRI-DWI). The patient should be admitted early and investigated. The use of rehabilitation services may be useful if available.
BACKGROUND: Surfers myelopathy can be a rapidly devastating disease and little is known surrounding the pathophysiology of the condition. Although the classical pattern of illness has been well reported, it has never been observed in a non-surfing setting. METHODS: A 51-year-old demolition worker presented with acute non-traumatic myelopathy. Clinical examination revealed sensory loss to the level of L2. T2-MRI and MRI-DWI revealed a hyperintense signal suggestive of an ischaemic event. A diagnosis of surfer's myelopathy was made and he was commenced on steroid therapy. RESULTS: Following steroid therapy and fluid management the patient was discharged after 6 days with minor anaethesia but significant overall neurological improvement. CONCLUSIONS: Diagnosis of SM requires a thorough history, clinical examination and imaging (MRI, MRI-DWI). The patient should be admitted early and investigated. The use of rehabilitation services may be useful if available.
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