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Literature DB >> 27694168

Evaluation of Prostate Cancer with 11C-Acetate PET/CT.

Claudio Spick¹, Ken Herrmann¹, Johannes Czernin².

Abstract

In this article, we will first describe the metabolic fate of 11C-acetate; then discuss its biodistribution in health and disease; and subsequently focus on its key clinical applications, the detection and localization of prostate cancer tissue in patients with primary or recurrent disease. Finally, we will discuss the potential role of 11C-acetate in the context of other prostate cancer imaging probes and non-radionuclide-based imaging approaches.

Entities: Chemical Disease Species

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Year: 2016 PMID： 27694168 DOI： 10.2967/jnumed.115.169599

Source DB: PubMed Journal: J Nucl Med ISSN： 0161-5505 Impact factor: 10.057

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Cited

14 in total

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5. Targeting ACSS2 with a Transition-State Mimetic Inhibits Triple-Negative Breast Cancer Growth.

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Evaluation of Prostate Cancer with 11C-Acetate PET/CT.

Review 1. Acetyl-CoA Synthetase 2 as a Therapeutic Target in Tumor Metabolism.

Review 2. Cancer cell metabolic plasticity in migration and metastasis.

Review 3. Tumour metabolism and its unique properties in prostate adenocarcinoma.

4. Propionic Acid-Based PET Imaging of Prostate Cancer.

5. Targeting ACSS2 with a Transition-State Mimetic Inhibits Triple-Negative Breast Cancer Growth.

6. Method to Development of PET Radiopharmaceutical for Cancer Imaging.

Review 7. Overcoming tumor hypoxia as a barrier to radiotherapy, chemotherapy and immunotherapy in cancer treatment.

8. When fat goes down, prostate cancer is on the ropes.

Review 9. Imaging Cancer Metabolism: Underlying Biology and Emerging Strategies.

10. Bispecific GRPR-Antagonistic Anti-PSMA/GRPR Heterodimer for PET and SPECT Diagnostic Imaging of Prostate Cancer.