Andreas Seidler1, Mandy Wagner2, Melanie Schubert2, Patrik Dröge2, Karin Römer3, Jörn Pons-Kühnemann3, Enno Swart4, Hajo Zeeb5, Janice Hegewald2. 1. Institute and Policlinic of Occupational and Social Medicine, TU Dresden, Faculty of Medicine Carl Gustav Carus, Dresden, Germany. Electronic address: Andreas.Seidler@mailbox.tu-dresden.de. 2. Institute and Policlinic of Occupational and Social Medicine, TU Dresden, Faculty of Medicine Carl Gustav Carus, Dresden, Germany. 3. Institute of Medical Informatics, Justus-Liebig-University Gießen, Germany. 4. Institute of Social Medicine and Health Economics, Otto-von-Guericke-University Magdeburg, Germany. 5. Department of Prevention and Evaluation, Leibniz-Institute for Prevention Research and Epidemiology-BIPS GmbH, Germany.
Abstract
BACKGROUND: Several studies point to an elevated risk for cardiovascular diseases induced by traffic noise. AIMS: We examined the association between aircraft, road traffic and railway noise and heart failure or hypertensive heart disease (HHD) in a large case-control study. METHODS: The study population consisted of individuals that were insured by three large statutory health insurance funds in the Rhine-Main area of Germany. Based on insurance claims and prescription data, 104,145 cases of heart failure or HHD diagnosed 2006-10 were identified and compared with 654,172 control subjects. Address-specific exposure to aircraft, road and railway traffic noise in 2005 was estimated. Odds Ratios were calculated using logistic regression analysis, adjusted for age, sex, local proportion of persons receiving unemployment benefits, and individual socioeconomic status (available for 39% of the individuals). RESULTS: A statistically significant linear exposure-risk relationship with heart failure or hypertensive heart disease was found for aircraft traffic noise (1.6% risk increase per 10dB increase in the 24-h continuous noise level; 95% CI 0.3-3.0%), road traffic noise (2.4% per 10dB; 95% CI 1.6-3.2%), and railway noise (3.1% per 10dB; 95% CI 2.2-4.1%). For individuals with 24-h continuous aircraft noise levels <40dB and nightly maximum aircraft noise levels exceeding 50dB six or more times, a significantly increased risk was observed. In general, risks of HHD were considerably higher than the risks of heart failure. CONCLUSIONS: Regarding the high prevalence of traffic noise from various sources, even low risk increases for frequent diseases are relevant for the population as a whole.
BACKGROUND: Several studies point to an elevated risk for cardiovascular diseases induced by traffic noise. AIMS: We examined the association between aircraft, road traffic and railway noise and heart failure or hypertensive heart disease (HHD) in a large case-control study. METHODS: The study population consisted of individuals that were insured by three large statutory health insurance funds in the Rhine-Main area of Germany. Based on insurance claims and prescription data, 104,145 cases of heart failure or HHD diagnosed 2006-10 were identified and compared with 654,172 control subjects. Address-specific exposure to aircraft, road and railway traffic noise in 2005 was estimated. Odds Ratios were calculated using logistic regression analysis, adjusted for age, sex, local proportion of persons receiving unemployment benefits, and individual socioeconomic status (available for 39% of the individuals). RESULTS: A statistically significant linear exposure-risk relationship with heart failure or hypertensive heart disease was found for aircraft traffic noise (1.6% risk increase per 10dB increase in the 24-h continuous noise level; 95% CI 0.3-3.0%), road traffic noise (2.4% per 10dB; 95% CI 1.6-3.2%), and railway noise (3.1% per 10dB; 95% CI 2.2-4.1%). For individuals with 24-h continuous aircraft noise levels <40dB and nightly maximum aircraft noise levels exceeding 50dB six or more times, a significantly increased risk was observed. In general, risks of HHD were considerably higher than the risks of heart failure. CONCLUSIONS: Regarding the high prevalence of traffic noise from various sources, even low risk increases for frequent diseases are relevant for the population as a whole.
Authors: Thomas Münzel; Mette Sørensen; Frank Schmidt; Erwin Schmidt; Sebastian Steven; Swenja Kröller-Schön; Andreas Daiber Journal: Antioxid Redox Signal Date: 2018-03-20 Impact factor: 8.401
Authors: Maria Foraster; Ikenna C Eze; Emmanuel Schaffner; Danielle Vienneau; Harris Héritier; Simon Endes; Franziska Rudzik; Laurie Thiesse; Reto Pieren; Christian Schindler; Arno Schmidt-Trucksäss; Mark Brink; Christian Cajochen; Jean Marc Wunderli; Martin Röösli; Nicole Probst-Hensch Journal: Environ Health Perspect Date: 2017-09-07 Impact factor: 9.031
Authors: Mette Sørensen; Olav Wendelboe Nielsen; Ahmad Sajadieh; Matthias Ketzel; Anne Tjønneland; Kim Overvad; Ole Raaschou-Nielsen Journal: Environ Health Perspect Date: 2017-09-26 Impact factor: 9.031
Authors: Junenette L Peters; Christopher D Zevitas; Susan Redline; Aaron Hastings; Natalia Sizov; Jaime E Hart; Jonathan I Levy; Christopher J Roof; Gregory A Wellenius Journal: Curr Epidemiol Rep Date: 2018-04-26
Authors: Michael Dierickx; Suzanne Verschraegen; Els Wierinck; Guy Willems; Astrid van Wieringen Journal: Int J Environ Res Public Health Date: 2021-05-24 Impact factor: 3.390
Authors: Miroslav Němec; Tomáš Gergeľ; Miloš Gejdoš; Anna Danihelová; Vojtěch Ondrejka Journal: Int J Environ Res Public Health Date: 2021-07-02 Impact factor: 3.390
Authors: Chloé Sieber; Martina S Ragettli; Mark Brink; Olaniyan Toyib; Roslyn Baatjies; Apolline Saucy; Nicole Probst-Hensch; Mohamed Aqiel Dalvie; Martin Röösli Journal: Int J Environ Res Public Health Date: 2017-10-20 Impact factor: 3.390