Literature DB >> 27630136

Experimentally Increasing the Compliance of Titin Through RNA Binding Motif-20 (RBM20) Inhibition Improves Diastolic Function In a Mouse Model of Heart Failure With Preserved Ejection Fraction.

Mei Methawasin1, Joshua G Strom1, Rebecca E Slater1, Vanessa Fernandez1, Chandra Saripalli1, Henk Granzier2.   

Abstract

BACKGROUND: Left ventricular (LV) stiffening contributes to heart failure with preserved ejection fraction (HFpEF), a syndrome with no effective treatment options. Increasing the compliance of titin in the heart has become possible recently through inhibition of the splicing factor RNA binding motif-20. Here, we investigated the effects of increasing the compliance of titin in mice with diastolic dysfunction.
METHODS: Mice in which the RNA recognition motif (RRM) of one of the RNA binding motif-20 alleles was floxed and that expressed the MerCreMer transgene under control of the αMHC promoter (referred to as cRbm20ΔRRM mice) were used. Mice underwent transverse aortic constriction (TAC) surgery and deoxycorticosterone acetate (DOCA) pellet implantation. RRM deletion in adult mice was triggered by injecting raloxifene (cRbm20ΔRRM-raloxifene), with dimethyl sulfoxide (DMSO)-injected mice (cRbm20ΔRRM-DMSO) as the control. Diastolic function was investigated with echocardiography and pressure-volume analysis; passive stiffness was studied in LV muscle strips and isolated cardiac myocytes before and after elimination of titin-based stiffness. Treadmill exercise performance was also studied. Titin isoform expression was evaluated with agarose gels.
RESULTS: cRbm20ΔRRM-raloxifene mice expressed large titins in the hearts, called supercompliant titin (N2BAsc), which, within 3 weeks after raloxifene injection, made up ≈45% of total titin. TAC/DOCA cRbm20ΔRRM-DMSO mice developed LV hypertrophy and a marked increase in LV chamber stiffness as shown by both pressure-volume analysis and echocardiography. LV chamber stiffness was normalized in TAC/DOCA cRbm20ΔRRM-raloxifene mice that expressed N2BAsc. Passive stiffness measurements on muscle strips isolated from the LV free wall revealed that extracellular matrix stiffness was equally increased in both groups of TAC/DOCA mice (cRbm20ΔRRM-DMSO and cRbm20ΔRRM-raloxifene). However, titin-based muscle stiffness was reduced in the mice that expressed N2BAsc (TAC/DOCAcRbm20ΔRRM-raloxifene). Exercise testing demonstrated significant improvement in exercise tolerance in TAC/DOCA mice that expressed N2BAsc.
CONCLUSIONS: Inhibition of the RNA binding motif-20-based titin splicing system upregulates compliant titins, which improves diastolic function and exercise tolerance in the TAC/DOCA model. Titin holds promise as a therapeutic target for heart failure with preserved ejection fraction.
© 2016 American Heart Association, Inc.

Entities:  

Keywords:  compliance; diastole; heart failure

Mesh:

Substances:

Year:  2016        PMID: 27630136      PMCID: PMC5069184          DOI: 10.1161/CIRCULATIONAHA.116.023003

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  46 in total

1.  2013 ACCF/AHA guideline for the management of heart failure: executive summary: a report of the American College of Cardiology Foundation/American Heart Association Task Force on practice guidelines.

Authors:  Clyde W Yancy; Mariell Jessup; Biykem Bozkurt; Javed Butler; Donald E Casey; Mark H Drazner; Gregg C Fonarow; Stephen A Geraci; Tamara Horwich; James L Januzzi; Maryl R Johnson; Edward K Kasper; Wayne C Levy; Frederick A Masoudi; Patrick E McBride; John J V McMurray; Judith E Mitchell; Pamela N Peterson; Barbara Riegel; Flora Sam; Lynne W Stevenson; W H Wilson Tang; Emily J Tsai; Bruce L Wilkoff
Journal:  Circulation       Date:  2013-06-05       Impact factor: 29.690

2.  Cardiac magnetic resonance-derived anatomy, scar, and dyssynchrony fused with fluoroscopy to guide LV lead placement in cardiac resynchronization therapy: a comparison with acute haemodynamic measures and echocardiographic reverse remodelling.

Authors:  Anoop K Shetty; Simon G Duckett; Matthew R Ginks; Yinglaing Ma; Manav Sohal; Julian Bostock; Stam Kapetanakis; Jagmeet P Singh; Kawal Rhode; Matthew Wright; Mark D O'Neill; Jaswinder S Gill; Gerald Carr-White; Reza Razavi; Christopher Aldo Rinaldi
Journal:  Eur Heart J Cardiovasc Imaging       Date:  2012-11-22       Impact factor: 6.875

3.  Mechanisms of diastolic dysfunction in heart failure with a preserved ejection fraction: If it's not one thing it's another.

Authors:  Martin M LeWinter; Markus Meyer
Journal:  Circ Heart Fail       Date:  2013-11       Impact factor: 8.790

4.  RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing.

Authors:  Wei Guo; Sebastian Schafer; Marion L Greaser; Michael H Radke; Martin Liss; Thirupugal Govindarajan; Henrike Maatz; Herbert Schulz; Shijun Li; Amanda M Parrish; Vita Dauksaite; Padmanabhan Vakeel; Sabine Klaassen; Brenda Gerull; Ludwig Thierfelder; Vera Regitz-Zagrosek; Timothy A Hacker; Kurt W Saupe; G William Dec; Patrick T Ellinor; Calum A MacRae; Bastian Spallek; Robert Fischer; Andreas Perrot; Cemil Özcelik; Kathrin Saar; Norbert Hubner; Michael Gotthardt
Journal:  Nat Med       Date:  2012-05       Impact factor: 53.440

5.  Relationship of reverse anatomical remodeling and ventricular arrhythmias after cardiac resynchronization.

Authors:  Steven M Markowitz; Jason M Lewen; Christopher J Wiggenhorn; William T Abraham; Kenneth M Stein; Sei Iwai; Bruce B Lerman
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6.  Effect of phosphodiesterase-5 inhibition on exercise capacity and clinical status in heart failure with preserved ejection fraction: a randomized clinical trial.

Authors:  Margaret M Redfield; Horng H Chen; Barry A Borlaug; Marc J Semigran; Kerry L Lee; Gregory Lewis; Martin M LeWinter; Jean L Rouleau; David A Bull; Douglas L Mann; Anita Deswal; Lynne W Stevenson; Michael M Givertz; Elizabeth O Ofili; Christopher M O'Connor; G Michael Felker; Steven R Goldsmith; Bradley A Bart; Steven E McNulty; Jenny C Ibarra; Grace Lin; Jae K Oh; Manesh R Patel; Raymond J Kim; Russell P Tracy; Eric J Velazquez; Kevin J Anstrom; Adrian F Hernandez; Alice M Mascette; Eugene Braunwald
Journal:  JAMA       Date:  2013-03-27       Impact factor: 56.272

Review 7.  Molecular and cellular basis for diastolic dysfunction.

Authors:  Loek van Heerebeek; Constantijn P M Franssen; Nazha Hamdani; Freek W A Verheugt; G Aernout Somsen; Walter J Paulus
Journal:  Curr Heart Fail Rep       Date:  2012-12

8.  Targeted deletion of titin N2B region leads to diastolic dysfunction and cardiac atrophy.

Authors:  Michael H Radke; Jun Peng; Yiming Wu; Mark McNabb; O Lynne Nelson; Henk Granzier; Michael Gotthardt
Journal:  Proc Natl Acad Sci U S A       Date:  2007-02-20       Impact factor: 11.205

9.  Truncation of titin's elastic PEVK region leads to cardiomyopathy with diastolic dysfunction.

Authors:  Henk L Granzier; Michael H Radke; Jun Peng; Dirk Westermann; O Lynne Nelson; Katharina Rost; Nicholas M P King; Qianli Yu; Carsten Tschöpe; Mark McNabb; Douglas F Larson; Siegfried Labeit; Michael Gotthardt
Journal:  Circ Res       Date:  2009-08-13       Impact factor: 17.367

10.  Mouse intact cardiac myocyte mechanics: cross-bridge and titin-based stress in unactivated cells.

Authors:  Nicholas M P King; Methajit Methawasin; Joshua Nedrud; Nicholas Harrell; Charles S Chung; Michiel Helmes; Henk Granzier
Journal:  J Gen Physiol       Date:  2011-01       Impact factor: 4.086

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  46 in total

1.  Could Modification of Titin Contribute to an Answer for Heart Failure With Preserved Ejection Fraction?

Authors:  Martin M LeWinter; Michael R Zile
Journal:  Circulation       Date:  2016-09-14       Impact factor: 29.690

Review 2.  Sirtuin 3, Endothelial Metabolic Reprogramming, and Heart Failure With Preserved Ejection Fraction.

Authors:  Heng Zeng; Jian-Xiong Chen
Journal:  J Cardiovasc Pharmacol       Date:  2019-10       Impact factor: 3.105

3.  Left ventricular function in patients with hypertrophic cardiomyopathy and its relation to myocardial fibrosis and exercise tolerance.

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Review 4.  Heart Failure With Preserved Ejection Fraction In Perspective.

Authors:  Marc A Pfeffer; Amil M Shah; Barry A Borlaug
Journal:  Circ Res       Date:  2019-05-24       Impact factor: 17.367

Review 5.  Role of mineralocorticoid receptor activation in cardiac diastolic dysfunction.

Authors:  Guanghong Jia; Yan Jia; James R Sowers
Journal:  Biochim Biophys Acta Mol Basis Dis       Date:  2016-10-29       Impact factor: 5.187

6.  Response by Methawasin and Granzier to Letter Regarding Article, "Experimentally Increasing the Compliance of Titin Through RNA Binding Motif-20 (RBM20) Inhibition Improves Diastolic Function in a Mouse Model of Heart Failure With Preserved Ejection Fraction".

Authors:  Mei Methawasin; Henk Granzier
Journal:  Circulation       Date:  2017-03-14       Impact factor: 29.690

7.  Softening the Stressed Giant Titin in Diabetes Mellitus.

Authors:  Mei Methawasin; Henk Granzier
Journal:  Circ Res       Date:  2018-07-20       Impact factor: 17.367

Review 8.  Role of Renin-Angiotensin-Aldosterone System Activation in Promoting Cardiovascular Fibrosis and Stiffness.

Authors:  Guanghong Jia; Annayya R Aroor; Michael A Hill; James R Sowers
Journal:  Hypertension       Date:  2018-09       Impact factor: 10.190

9.  The structural basis of alpha-tropomyosin linked (Asp230Asn) familial dilated cardiomyopathy.

Authors:  M L Lynn; L Tal Grinspan; T A Holeman; J Jimenez; J Strom; J C Tardiff
Journal:  J Mol Cell Cardiol       Date:  2017-06-07       Impact factor: 5.000

Review 10.  Novex-3, the tiny titin of muscle.

Authors:  Dalma Kellermayer; John E Smith; Henk Granzier
Journal:  Biophys Rev       Date:  2017-04-07
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