Literature DB >> 27619208

Assessment of tobacco specific nitrosamines (TSNAs) in oral fluid as biomarkers of cancer risk: A population-based study.

Raúl Pérez-Ortuño1, Jose M Martínez-Sánchez2, Marcela Fu3, Montse Ballbè4, Núria Quirós5, Esteve Fernández6, José A Pascual7.   

Abstract

BACKGROUND: Smoke-free laws are expected to reduce smoking habits and exposure to secondhand smoke. The objective of this study was the measurement of tobacco specific carcinogens (TSNAs) in oral fluid to assess the most suitable biomarker of cancer risk associated with tobacco smoke.
METHODS: TSNAs, N'-nitrosonornicotine (NNN), 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanol (NNAL), as well as nicotine and cotinine were measured in oral fluid samples from 166 smokers and 532 non-smokers of the adult population of Barcelona, Spain. A simple method with an alkaline single liquid-liquid extraction with dichloromethane/isopropanol was used and lower limits of quantification for cotinine, NNN, NNK and NNAL were set at 0.10ng/mL, 1.0, 2.0 and 0.50pg/mL respectively. The NNN/cotinine ratio was also calculated.
RESULTS: NNN was the most abundant TSNA present in oral fluid with a significant difference between smokers and non-smokers (mean concentrations of 118 and 5.3pg/mL, respectively, p<0.001). NNK and NNAL were detectable in fewer samples. NNN and cotinine concentrations had a moderate correlation within both groups (Spearman's rank correlation coefficient of 0.312, p<0.001 in smokers and 0.279, p=0.022 in non-smokers). NNN/cotinine ratio was significantly higher (p<0.001) in non-smokers than in smokers, in line with equivalent findings for the NNAL/cotinine ratio in urine.
CONCLUSIONS: TSNAs are detectable in oral fluid of smokers and non-smokers. NNN is the most abundant, in line with its association with esophageal and oral cavity cancers. The NNN/cotinine ratio confirms the relative NNN increase in second hand smoke. Findings provide a new oral fluid biomarker of cancer risk associated with exposure to tobacco smoke.
Copyright © 2016 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  NNN; Saliva; TSNA; Tobacco biomarker; Tobacco carcinogen

Mesh:

Substances:

Year:  2016        PMID: 27619208     DOI: 10.1016/j.envres.2016.08.036

Source DB:  PubMed          Journal:  Environ Res        ISSN: 0013-9351            Impact factor:   6.498


  5 in total

Review 1.  Metabolism and DNA Adduct Formation of Tobacco-Specific N-Nitrosamines.

Authors:  Yupeng Li; Stephen S Hecht
Journal:  Int J Mol Sci       Date:  2022-05-04       Impact factor: 6.208

2.  Metabolites of Tobacco- and E-Cigarette-Related Nitrosamines Can Drive Cu2+-Mediated DNA Oxidation.

Authors:  Rumasha N T Kankanamage; Abhisek Brata Ghosh; Di Jiang; Karmel Gkika; Tia Keyes; Laura A Achola; Steven Suib; James F Rusling
Journal:  Chem Res Toxicol       Date:  2020-08-04       Impact factor: 3.739

Review 3.  Advances of Salivary Proteomics in Oral Squamous Cell Carcinoma (OSCC) Detection: An Update.

Authors:  Rabia Sannam Khan; Zohaib Khurshid; Shazia Akhbar; Syed Faraz Moin
Journal:  Proteomes       Date:  2016-12-15

Review 4.  Biomarkers of Exposure to Secondhand and Thirdhand Tobacco Smoke: Recent Advances and Future Perspectives.

Authors:  Sònia Torres; Carla Merino; Beatrix Paton; Xavier Correig; Noelia Ramírez
Journal:  Int J Environ Res Public Health       Date:  2018-11-29       Impact factor: 3.390

5.  Permanence of Modifications in Oral Microcirculation in Ex-Smokers.

Authors:  Giuseppe A Scardina; Massimiliano Messina; Dario Melilli; Enzo Cumbo; Francesco Carini; Giovanni Tomasello; Pietro Messina
Journal:  Med Sci Monit       Date:  2019-01-30
  5 in total

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