Literature DB >> 27616009

Utility of high density lipoprotein particle concentration in predicting future major adverse cardiovascular events among patients undergoing angiography.

Heidi T May1, Jeffrey L Anderson2, Deborah A Winegar3, Jeffrey Rollo4, Margery A Connelly3, James D Otvos3, Joseph B Muhlestein2.   

Abstract

BACKGROUND: HDL-C is recognized to be inversely associated with cardiovascular (CV) risk. However, attenuation of the association of HDL-C with CV risk may occur after adjustment for other lipoprotein parameters and in various disease states, especially in the setting of acute coronary syndrome (ACS). Recently, the number of HDL particles (HDL-P) has been suggested to improve CV risk prediction. METHODS AND
RESULTS: Patients (n=2999) in the Intermountain Heart Collaborative Study who underwent angiography and had lipoprotein particle measurements determined by nuclear magnetic resonance (NMR) spectroscopy were studied. Multivariable Cox hazard regression was utilized to evaluate the association of HDL-C, HDL-P, and HDL-P subclasses with future major adverse CV events (MACE: death, myocardial infarction, heart failure, and stroke). Patients averaged 64±12years, 66% male, 26% diabetic, and 42% ACS. At angiography, 65% of patients were diagnosed with coronary artery disease (CAD). HDL-C and HDL-P averaged 41±13mg/dL and 28±8μmol/L, respectively. HDL-P (HR=0.903, p=0.001), but not HDL-C (HR=0.947, p=0.102) was significantly associated with MACE. In a model that included all HDL-P subclasses, both small (HR=0.862, p<0.0001) and medium (HR=0.922, p=0.020) were associated with CV risk, but not large HDL-P (HR=1.0042, p=0.185). Small HDL-P continued to be associated with all of the individual components of MACE, but not stroke.
CONCLUSION: In this study of patients undergoing angiography, HDL-P was a strong, independent predictor of future MACE, with the smaller HDL-P accounting for this association.
Copyright © 2016 The Canadian Society of Clinical Chemists. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Cardiovascular risk; Lipids; Outcomes

Mesh:

Substances:

Year:  2016        PMID: 27616009     DOI: 10.1016/j.clinbiochem.2016.09.004

Source DB:  PubMed          Journal:  Clin Biochem        ISSN: 0009-9120            Impact factor:   3.281


  2 in total

1.  Association of long-term PM2.5 exposure with traditional and novel lipid measures related to cardiovascular disease risk.

Authors:  Laura A McGuinn; Alexandra Schneider; Robert W McGarrah; Cavin Ward-Caviness; Lucas M Neas; Qian Di; Joel Schwartz; Elizabeth R Hauser; William E Kraus; Wayne E Cascio; David Diaz-Sanchez; Robert B Devlin
Journal:  Environ Int       Date:  2018-11-13       Impact factor: 9.621

2.  Cardiovascular disease risk factor responses to a type 2 diabetes care model including nutritional ketosis induced by sustained carbohydrate restriction at 1 year: an open label, non-randomized, controlled study.

Authors:  Nasir H Bhanpuri; Sarah J Hallberg; Paul T Williams; Amy L McKenzie; Kevin D Ballard; Wayne W Campbell; James P McCarter; Stephen D Phinney; Jeff S Volek
Journal:  Cardiovasc Diabetol       Date:  2018-05-01       Impact factor: 9.951

  2 in total

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