Literature DB >> 27589992

Insulin resistance, glucagon-like peptide-1 and factors influencing glucose homeostasis in neonates.

Emma R Salis1, David M Reith2, Benjamin J Wheeler2, Roland S Broadbent2, Natalie J Medlicott1.   

Abstract

OBJECTIVES: To explore the relationships between postmenstrual age (PMA), insulin, C-peptide, glucagon and blood glucose concentrations (BGCs) in preterm and term neonates. To compare glucagon-like peptide-1 (GLP-1) concentrations in fed versus never-fed neonates.
DESIGN: Observational.
SETTING: Dunedin Hospital Neonatal Intensive Care Unit, New Zealand. PATIENTS: Term or preterm euglycaemic neonates (102) receiving routine blood tests (343 samples).
INTERVENTIONS: None: plasma was obtained from surplus samples from routine clinical care. MAIN OUTCOME MEASURES: Insulin, C-peptide, GLP-1 and glucagon concentrations were measured in temporal association with BGC.
RESULTS: Insulin and C-peptide concentrations were elevated in very preterm infants (PMA≤32 weeks) and decreased to term; this relationship persisted when BGCs were accounted for. Generalised linear mixed models showed that insulin:C-peptide ratio and insulin:BGC ratio decreased significantly with increasing PMA (p<0.001). GLP-1 increased following initial oral feeds regardless of PMA (p<0.001).
CONCLUSION: Preterm neonates exhibit insulin resistance in the absence of hyperglycaemia. Enteral feeds result in an increase in GLP-1. These factors are likely to contribute to the increased risk of hyperglycaemia in premature neonates (PMA<32 weeks). Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/.

Entities:  

Keywords:  GLP-1; glucagon; hyperglycaemia; insulin; neonate

Mesh:

Substances:

Year:  2016        PMID: 27589992     DOI: 10.1136/archdischild-2015-309174

Source DB:  PubMed          Journal:  Arch Dis Child Fetal Neonatal Ed        ISSN: 1359-2998            Impact factor:   5.747


  4 in total

1.  Prematurity blunts the feeding-induced stimulation of translation initiation signaling and protein synthesis in muscle of neonatal piglets.

Authors:  Jane K Naberhuis; Agus Suryawan; Hanh V Nguyen; Adriana Hernandez-Garcia; Stephanie M Cruz; Patricio E Lau; Oluyinka O Olutoye; Barbara Stoll; Douglas G Burrin; Marta L Fiorotto; Teresa A Davis
Journal:  Am J Physiol Endocrinol Metab       Date:  2019-09-10       Impact factor: 4.310

2.  Prematurity at less than 24 weeks of gestation is a risk for prolonged hyperglycemia in extremely low-birth weight infants.

Authors:  Takeru Yamauchi; Masatoshi Imamura; Kei Takasawa; Keisuke Nakajima; Ryuichi Nakagawa; Maki Gau; Manabu Sugie; Atsuko Taki; Masahiko Kawai; Kenichi Kashimada; Tomohiro Morio
Journal:  Endocrine       Date:  2020-07-02       Impact factor: 3.633

Review 3.  Epigenetic Programming and Fetal Metabolic Programming.

Authors:  Ziqiang Zhu; Fang Cao; Xiaozhong Li
Journal:  Front Endocrinol (Lausanne)       Date:  2019-12-03       Impact factor: 5.555

4.  Hyperglycaemic preterm neonates exhibit insulin resistance and low insulin production.

Authors:  Emma R Salis; David M Reith; Benjamin J Wheeler; Roland S Broadbent; Natalie J Medlicott
Journal:  BMJ Paediatr Open       Date:  2017-09-05
  4 in total

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