Literature DB >> 27552106

The LG/J murine strain exhibits near-normal tendon biomechanical properties following a full-length central patellar tendon defect.

Jessica R Arble1, Andrea L Lalley2, Nathaniel A Dyment3, Pujan Joshi4, Dong-Guk Shin4, Cynthia Gooch1, Brian Grawe5, David Rowe3, Jason T Shearn1.   

Abstract

PURPOSE OF THE STUDY: Identifying biological success criteria is needed to improve therapies, and one strategy for identifying them is to analyze the RNA transcriptome for successful and unsuccessful models of tendon healing. We have characterized the MRL/MpJ murine strain and found improved mechanical outcomes following a central patellar tendon (PT) injury. In this study, we evaluate the healing of the LG/J murine strain, which comprises 75% of the MRL/MpJ background, to determine if the LG/J also exhibits improved biomechanical properties following injury and to determine differentially expressed transcription factors across the C57BL/6, MRL/MpJ and the LG/J strains during the early stages of healing.
MATERIALS AND METHODS: A full-length, central PT defect was created in 16-20 week old MRL/MpJ, LG/J, and C57BL/6 murine strains. Mechanical properties were assessed at 2, 5, and 8 weeks post surgery. Transcriptomic expression was assessed at 3, 7, and 14 days following injury using a novel clustering software program to evaluate differential expression of transcription factors.
RESULTS: Average LG/J structural properties improved to 96.7% and 97.2% of native LG/J PT stiffness and ultimate load by 8 weeks post surgery, respectively. We found the LG/J responded by increasing expression of transcription factors implicated in the inflammatory response and collagen fibril organization.
CONCLUSIONS: The LG/J strain returns to normal structural properties by 8 weeks, with steadily increasing properties at each time point. Future work will characterize the cell populations responding to injury and investigate the role of the differentially expressed transcription factors during healing.

Entities:  

Keywords:  Biomechanical properties; mouse model; tendon healing; transcription factors; transcriptome

Mesh:

Year:  2016        PMID: 27552106     DOI: 10.1080/03008207.2016.1213247

Source DB:  PubMed          Journal:  Connect Tissue Res        ISSN: 0300-8207            Impact factor:   3.417


  2 in total

1.  Macrophage depletion impairs neonatal tendon regeneration.

Authors:  Kristen L Howell; Deepak A Kaji; Thomas M Li; Angela Montero; Kenji Yeoh; Philip Nasser; Alice H Huang
Journal:  FASEB J       Date:  2021-06       Impact factor: 5.834

2.  Novel Model of Tendon Regeneration Reveals Distinct Cell Mechanisms Underlying Regenerative and Fibrotic Tendon Healing.

Authors:  Kristen Howell; Chun Chien; Rebecca Bell; Damien Laudier; Sara F Tufa; Douglas R Keene; Nelly Andarawis-Puri; Alice H Huang
Journal:  Sci Rep       Date:  2017-03-23       Impact factor: 4.379

  2 in total

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