Literature DB >> 27526097

Intracranial undifferentiated malign neuroglial tumor in Smith-Lemli-Opitz syndrome: A theory of a possible predisposing factor for primary brain tumors via a case report.

Ayfer Aslan1, Alp Ozgun Borcek2, Selma Pamukcuoglu3, M Kemali Baykaner2.   

Abstract

BACKGROUND: Smith-Lemli-Opitz Syndrome (SLOS) is a rare hereditary autosomal recessive disorder with broken cholesterol synthesis causing by 7-dehydrocholesterol reductase deficiency. Although the clinical features and pathogenesis is well-defined, it is unknown whether there is a relationship between SLOS and neoplastic processes, especially brain neoplasms.
PURPOSE: We aimed to attract the attentions to any possibility of relation between SLOS and intracranial tumor development via a pediatric case with both intracranial high-grade neuroglial tumor and SLOS, and thus to contribute an additional data to the literature on togetherness of these two clinical conditions.
METHOD: In our clinic, we experienced an interesting case of a 10-year-old child with both SLOS and huge brain tumor as rarely seen. Here, we reviewed the features and pathophysiology of SLOS and brain tumors via this case.
RESULTS: The patient was operated in our clinic, after, his brain tumor had been diagnosed, and his histopathology was resulted in undifferentiated malignant neuroglial WHO grade 4 tumor.
CONCLUSION: According to current literature, our case is the first report on coexisting of SLOS and intracranial undifferentiated malignant neuroglial tumor. Common pathways like impaired sonic hedgehog (Shh) signaling pathway may be considered for pathogenesis of a probable link between SLOS and brain tumors in further studies.

Entities:  

Keywords:  Brain surgery; Intracranial undifferentiated malignant tumor; Smith-Lemli-Opitz syndrome; Sonic Hedgehog signaling pathway

Mesh:

Substances:

Year:  2016        PMID: 27526097     DOI: 10.1007/s00381-016-3214-z

Source DB:  PubMed          Journal:  Childs Nerv Syst        ISSN: 0256-7040            Impact factor:   1.475


  31 in total

1.  Sonic hedgehog participates in craniofacial morphogenesis and is down-regulated by teratogenic doses of retinoic acid.

Authors:  J A Helms; C H Kim; D Hu; R Minkoff; C Thaller; G Eichele
Journal:  Dev Biol       Date:  1997-07-01       Impact factor: 3.582

Review 2.  The Hedgehog and Wnt signalling pathways in cancer.

Authors:  J Taipale; P A Beachy
Journal:  Nature       Date:  2001-05-17       Impact factor: 49.962

3.  Generation of cancerous neural stem cells forming glial tumor by oncogenic stimulation.

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Journal:  Cell Cycle       Date:  2016-05-18       Impact factor: 4.534

5.  Dispatched, a novel sterol-sensing domain protein dedicated to the release of cholesterol-modified hedgehog from signaling cells.

Authors:  R Burke; D Nellen; M Bellotto; E Hafen; K A Senti; B J Dickson; K Basler
Journal:  Cell       Date:  1999-12-23       Impact factor: 41.582

Review 6.  The Smith-Lemli-Opitz syndrome.

Authors:  R I Kelley; R C Hennekam
Journal:  J Med Genet       Date:  2000-05       Impact factor: 6.318

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Authors:  R L Kelley; E Roessler; R C Hennekam; G L Feldman; K Kosaki; M C Jones; J C Palumbos; M Muenke
Journal:  Am J Med Genet       Date:  1996-12-30

8.  Widespread requirement for Hedgehog ligand stimulation in growth of digestive tract tumours.

Authors:  David M Berman; Sunil S Karhadkar; Anirban Maitra; Rocio Montes De Oca; Meg R Gerstenblith; Kimberly Briggs; Antony R Parker; Yutaka Shimada; James R Eshleman; D Neil Watkins; Philip A Beachy
Journal:  Nature       Date:  2003-09-14       Impact factor: 49.962

9.  The p53 inhibitor MDM2 facilitates Sonic Hedgehog-mediated tumorigenesis and influences cerebellar foliation.

Authors:  Reem Malek; Jennifer Matta; Natalie Taylor; Mary Ellen Perry; Susan M Mendrysa
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Authors:  Lukas Tamayo-Orrego; Shannon M Swikert; Frédéric Charron
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