Literature DB >> 27443814

Associations between maternal periconceptional exposure to secondhand tobacco smoke and major birth defects.

Adrienne T Hoyt1, Mark A Canfield2, Paul A Romitti3, Lorenzo D Botto4, Marlene T Anderka5, Sergey V Krikov4, Morgan K Tarpey2, Marcia L Feldkamp4.   

Abstract

BACKGROUND: While associations between secondhand smoke and a few birth defects (namely, oral clefts and neural tube defects) have been noted in the scientific literature, to our knowledge, there is no single or comprehensive source of population-based information on its associations with a range of birth defects among nonsmoking mothers.
OBJECTIVE: We utilized data from the National Birth Defects Prevention Study, a large population-based multisite case-control study, to examine associations between maternal reports of periconceptional exposure to secondhand smoke in the household or workplace/school and major birth defects. STUDY
DESIGN: The multisite National Birth Defects Prevention Study is the largest case-control study of birth defects to date in the United States. We selected cases from birth defect groups having >100 total cases, as well as all nonmalformed controls (10,200), from delivery years 1997 through 2009; 44 birth defects were examined. After excluding cases and controls from multiple births and whose mothers reported active smoking or pregestational diabetes, we analyzed data on periconceptional secondhand smoke exposure-encompassing the period 1 month prior to conception through the first trimester. For the birth defect craniosynostosis, we additionally examined the effect of exposure in the second and third trimesters as well due to the potential sensitivity to teratogens for this defect throughout pregnancy. Covariates included in all final models of birth defects with ≥5 exposed mothers were study site, previous live births, time between estimated date of delivery and interview date, maternal age at estimated date of delivery, race/ethnicity, education, body mass index, nativity, household income divided by number of people supported by this income, periconceptional alcohol consumption, and folic acid supplementation. For each birth defect examined, we used logistic regression analyses to estimate both crude and adjusted odds ratios and 95% confidence intervals for both isolated and total case groups for various sources of exposure (household only; workplace/school only; household and workplace/school; household or workplace/school).
RESULTS: The prevalence of secondhand smoke exposure only across all sources ranged from 12.9-27.8% for cases and 14.5-15.8% for controls. The adjusted odds ratios for any vs no secondhand smoke exposure in the household or workplace/school and isolated birth defects were significantly elevated for neural tube defects (anencephaly: adjusted odds ratio, 1.66; 95% confidence interval, 1.22-2.25; and spina bifida: adjusted odds ratio, 1.49; 95% confidence interval, 1.20-1.86); orofacial clefts (cleft lip without cleft palate: adjusted odds ratio, 1.41; 95% confidence interval, 1.10-1.81; cleft lip with or without cleft palate: adjusted odds ratio, 1.24; 95% confidence interval, 1.05-1.46; cleft palate alone: adjusted odds ratio, 1.31; 95% confidence interval, 1.06-1.63); bilateral renal agenesis (adjusted odds ratio, 1.99; 95% confidence interval, 1.05-3.75); amniotic band syndrome-limb body wall complex (adjusted odds ratio, 1.66; 95% confidence interval, 1.10-2.51); and atrial septal defects, secundum (adjusted odds ratio, 1.37; 95% confidence interval, 1.09-1.72). There were no significant inverse associations observed.
CONCLUSION: Additional studies replicating the findings are needed to better understand the moderate positive associations observed between periconceptional secondhand smoke and several birth defects in this analysis. Increased odds ratios resulting from chance (eg, multiple comparisons) or recall bias cannot be ruled out.
Copyright © 2016 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  birth defects; congenital defects; congenital heart defects; environmental tobacco smoke; malformations; passive smoking; secondhand smoke

Mesh:

Substances:

Year:  2016        PMID: 27443814     DOI: 10.1016/j.ajog.2016.07.022

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN: 0002-9378            Impact factor:   8.661


  18 in total

1.  Rates of exposure to environmental tobacco smoke from various indoor environments among US children and nonsmoker adolescents and adults.

Authors:  Ram B Jain
Journal:  Environ Sci Pollut Res Int       Date:  2018-04-07       Impact factor: 4.223

2.  Potential risk factors for Ebstein anomaly, National Birth Defects Prevention Study, 1997-2011.

Authors:  Karrie F Downing; Tiffany Riehle-Colarusso; Suzanne M Gilboa; Angela E Lin; Matthew E Oster; Sarah C Tinker; Sherry L Farr
Journal:  Cardiol Young       Date:  2019-06-04       Impact factor: 1.093

Review 3.  [Cleft lip and palate].

Authors:  A Voigt; R J Radlanski; N Sarioglu; G Schmidt
Journal:  Pathologe       Date:  2017-07       Impact factor: 1.011

4.  Pregestational type 2 diabetes mellitus induces cardiac hypertrophy in the murine embryo through cardiac remodeling and fibrosis.

Authors:  Xue Lin; Penghua Yang; E Albert Reece; Peixin Yang
Journal:  Am J Obstet Gynecol       Date:  2017-04-13       Impact factor: 8.661

Review 5.  Gene/environment interactions in craniosynostosis: A brief review.

Authors:  E L Durham; R N Howie; J J Cray
Journal:  Orthod Craniofac Res       Date:  2017-06       Impact factor: 1.826

6.  Association between maternal exposure to tobacco, presence of TGFA gene, and the occurrence of oral clefts. A case control study.

Authors:  Mohammed Junaid; M B Aswath Narayanan; D Jayanthi; S G Ramesh Kumar; A Leena Selvamary
Journal:  Clin Oral Investig       Date:  2017-03-16       Impact factor: 3.573

Review 7.  Epigenetics and Congenital Heart Diseases.

Authors:  Léa Linglart; Damien Bonnet
Journal:  J Cardiovasc Dev Dis       Date:  2022-06-09

8.  Maternal Smoking and Congenital Heart Defects, National Birth Defects Prevention Study, 1997-2011.

Authors:  Elijah H Bolin; Yevgeniya Gokun; Paul A Romitti; Sarah C Tinker; April D Summers; Paula K Roberson; Charlotte A Hobbs; Sadia Malik; Lorenzo D Botto; Wendy N Nembhard
Journal:  J Pediatr       Date:  2021-09-08       Impact factor: 4.406

9.  Correlation between TGF-β2/3 promoter DNA methylation and Smad signaling during palatal fusion induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin.

Authors:  Yao Chen; Xiaozhuan Liu; Xinxin Liu; Lingling Cui; Zhidong He; Zhan Gao; Limin Liu; Zhitao Li; Zhongxiao Wan; Zengli Yu
Journal:  Exp Biol Med (Maywood)       Date:  2021-05-30

Review 10.  Smoking and pregnancy: Epigenetics and developmental origins of the metabolic syndrome.

Authors:  John M Rogers
Journal:  Birth Defects Res       Date:  2019-07-16       Impact factor: 2.661

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