Literature DB >> 27394401

De-escalation versus continuation of empirical antimicrobial therapy in community-acquired pneumonia.

Hayato Yamana1, Hiroki Matsui2, Takashi Tagami3, Junko Hirashima4, Kiyohide Fushimi5, Hideo Yasunaga2.   

Abstract

OBJECTIVES: To compare mortality between de-escalation and continued empirical treatment in patients with community-acquired pneumonia.
METHODS: Using a nationwide administrative database, we identified adult patients with community-acquired pneumonia caused by Streptococcus pneumoniae, other streptococci, Haemophilus influenzae, Klebsiella pneumoniae, or Escherichia coli (n = 10,231) or of unknown etiology (n = 8247), discharged between July 2010 and March 2013. De-escalation was determined by the spectrum and number of antimicrobials at day 4. We used propensity score matching to obtain 489 pairs of de-escalation and continuation groups among pathogen-identified patients and 278 pairs among culture-negative patients to compare mortalities.
RESULTS: In the pathogen-identified patients, de-escalation was noninferior to continuation in 15-day mortality [5.3% in de-escalation versus 4.3% in continuation, a difference of 1.0% (95% confidence interval, -1.7% to 3.7%)] and in-hospital mortality [8.0% in de-escalation versus 8.8% in continuation, a difference of -0.8% (95% confidence interval, -4.3% to 2.7%)]. In the culture-negative cases, de-escalation was noninferior to continuation in terms of 15-day mortality but not in terms of in-hospital mortality.
CONCLUSIONS: Among patients with community-acquired pneumonia of specific etiology, de-escalation was noninferior to continuation of empirical treatment, suggesting that de-escalation is a safe strategy and supporting current recommendations. Safety of de-escalation in culture-negative cases is questionable.
Copyright © 2016 The British Infection Association. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Anti-infective agents; Bacterial pneumonia; Drug resistance

Mesh:

Substances:

Year:  2016        PMID: 27394401     DOI: 10.1016/j.jinf.2016.07.001

Source DB:  PubMed          Journal:  J Infect        ISSN: 0163-4453            Impact factor:   6.072


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