Literature DB >> 27392540

Selective Disruption of Respiratory Supercomplexes as a New Strategy to Suppress Her2high Breast Cancer.

Katerina Rohlenova1, Karishma Sachaphibulkij2, Jan Stursa2,3,4, Ayenachew Bezawork-Geleta2, Jan Blecha1, Berwini Endaya2, Lukas Werner4, Jiri Cerny1, Renata Zobalova1,2, Jacob Goodwin2, Tomas Spacek5, Elham Alizadeh Pesdar2, Bing Yan2, Maria Nga Nguyen2, Magdalena Vondrusova1, Margaryta Sobol6, Petr Jezek5, Pavel Hozak6, Jaroslav Truksa1, Jakub Rohlena1, Lan-Feng Dong2, Jiri Neuzil1,2.   

Abstract

AIMS: Expression of the HER2 oncogene in breast cancer is associated with resistance to treatment, and Her2 may regulate bioenergetics. Therefore, we investigated whether disruption of the electron transport chain (ETC) is a viable strategy to eliminate Her2high disease.
RESULTS: We demonstrate that Her2high cells and tumors have increased assembly of respiratory supercomplexes (SCs) and increased complex I-driven respiration in vitro and in vivo. They are also highly sensitive to MitoTam, a novel mitochondrial-targeted derivative of tamoxifen. Unlike tamoxifen, MitoTam efficiently suppresses experimental Her2high tumors without systemic toxicity. Mechanistically, MitoTam inhibits complex I-driven respiration and disrupts respiratory SCs in Her2high background in vitro and in vivo, leading to elevated reactive oxygen species production and cell death. Intriguingly, higher sensitivity of Her2high cells to MitoTam is dependent on the mitochondrial fraction of Her2. INNOVATION: Oncogenes such as HER2 can restructure ETC, creating a previously unrecognized therapeutic vulnerability exploitable by SC-disrupting agents such as MitoTam.
CONCLUSION: We propose that the ETC is a suitable therapeutic target in Her2high disease. Antioxid. Redox Signal. 26, 84-103.

Entities:  

Keywords:  HER2; breast cancer; mitochondria; mitochondrially targeted tamoxifen; respirasome

Mesh:

Substances:

Year:  2016        PMID: 27392540      PMCID: PMC5206771          DOI: 10.1089/ars.2016.6677

Source DB:  PubMed          Journal:  Antioxid Redox Signal        ISSN: 1523-0864            Impact factor:   8.401


  60 in total

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2.  Mitochondrial targeting of vitamin E succinate enhances its pro-apoptotic and anti-cancer activity via mitochondrial complex II.

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Authors:  L-F Dong; P Low; J C Dyason; X-F Wang; L Prochazka; P K Witting; R Freeman; E Swettenham; K Valis; J Liu; R Zobalova; J Turanek; D R Spitz; F E Domann; I E Scheffler; S J Ralph; J Neuzil
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  33 in total

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Journal:  Cell Metab       Date:  2018-11-15       Impact factor: 27.287

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Review 4.  Active mitochondrial respiration in cancer: a target for the drug.

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Review 5.  Mitochondria as a Novel Target for Cancer Chemoprevention: Emergence of Mitochondrial-targeting Agents.

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7.  Mitochondrial Respiration in Human Colorectal and Breast Cancer Clinical Material Is Regulated Differently.

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8.  Cancer Cell Mitochondria Targeting by Pancratistatin Analogs is Dependent on Functional Complex II and III.

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Review 9.  Mitochondrial respiratory supercomplexes in mammalian cells: structural versus functional role.

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Authors:  Anna Yu Spivak; Darya A Nedopekina; Rinat R Gubaidullin; Mikhail V Dubinin; Konstantin N Belosludtsev
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