Literature DB >> 27353469

Sequential steps of daptomycin resistance in Enterococcus faecium and reversion to hypersusceptibility through IS-mediated inactivation of the liaFSR operon.

Clara Sinel1, Thibaud Cosquer1, Michel Auzou2, Didier Goux3, Jean-Christophe Giard1, Vincent Cattoir4.   

Abstract

OBJECTIVES: To improve understanding of mechanisms of daptomycin resistance and to dissect the genetic basis of reversion to daptomycin hypersusceptibility in Enterococcus faecium.
METHODS: Daptomycin-resistant mutants (Mut4, Mut8, Mut16, Mut32, Mut64 and Mut128 with MICs from 4 to 128 mg/L) were obtained in vitro from E. faecium strain Aus0004 (MIC at 2 mg/L). The entire genome sequences of Mut64 and Mut128 were determined as well as those of liaFSR and cls genes for other mutants and corresponding revertants (named Rev4 to Rev128). The study of daptomycin resistance stability was performed without any selective pressure. The expression of liaF, liaS and liaR genes was quantified by quantitative RT-PCR.
RESULTS: By comparative genomic analysis, substitutions Asn13Ser in cls and Gly92Asp in liaS were identified in Mut64 and Mut128. Only the liaS mutation was found in Mut16 and Mut32 while Mut4 and Mut8 were devoid of any mutation. After 15 days, all mutants except Mut4 reverted to daptomycin hypersusceptibility (MICs from 0.12 to 0.25 mg/L). In all revertants (except Rev4 and Rev8), an IS was found in the liaFSR operon with a dramatic decrease of its expression: IS66 in the promoter region of liaF (Rev16 and Rev64), IS30 in liaR (Rev32) and IS982 in liaF (Rev128).
CONCLUSIONS: We demonstrated the stepwise and sequential acquisition of mutations in liaS and in cls leading to daptomycin resistance in E. faecium, and the instability of daptomycin resistance as well as the role of liaFSR inactivation in reversion to daptomycin hypersusceptibility.
© The Author 2016. Published by Oxford University Press on behalf of the British Society for Antimicrobial Chemotherapy. All rights reserved. For Permissions, please e-mail: journals.permissions@oup.com.

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Year:  2016        PMID: 27353469     DOI: 10.1093/jac/dkw229

Source DB:  PubMed          Journal:  J Antimicrob Chemother        ISSN: 0305-7453            Impact factor:   5.790


  4 in total

1.  A mutation in the glycosyltransferase gene lafB causes daptomycin hypersusceptibility in Enterococcus faecium.

Authors:  Suelen S Mello; Daria Van Tyne; Francois Lebreton; Simone Q Silva; Mara C L Nogueira; Michael S Gilmore; Ilana L B C Camargo
Journal:  J Antimicrob Chemother       Date:  2020-01-01       Impact factor: 5.790

2.  RelA Mutant Enterococcus faecium with Multiantibiotic Tolerance Arising in an Immunocompromised Host.

Authors:  Erin S Honsa; Vaughn S Cooper; Mohammed N Mhaissen; Matthew Frank; Jessica Shaker; Amy Iverson; Jeffrey Rubnitz; Randall T Hayden; Richard E Lee; Charles O Rock; Elaine I Tuomanen; Joshua Wolf; Jason W Rosch
Journal:  MBio       Date:  2017-01-03       Impact factor: 7.867

3.  Evolution and mutations predisposing to daptomycin resistance in vancomycin-resistant Enterococcus faecium ST736 strains.

Authors:  Guiqing Wang; Fan Yu; Henry Lin; Karthikeyan Murugesan; Weihua Huang; Andrew G Hoss; Abhay Dhand; Leslie Y Lee; Jian Zhuge; Changhong Yin; Marisa Montecalvo; Nevenka Dimitrova; John T Fallon
Journal:  PLoS One       Date:  2018-12-21       Impact factor: 3.240

4.  Small RNAs in vancomycin-resistant Enterococcus faecium involved in daptomycin response and resistance.

Authors:  Clara Sinel; Yoann Augagneur; Mohamed Sassi; Julie Bronsard; Margherita Cacaci; François Guérin; Maurizio Sanguinetti; Pierrick Meignen; Vincent Cattoir; Brice Felden
Journal:  Sci Rep       Date:  2017-09-11       Impact factor: 4.379

  4 in total

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