| Literature DB >> 27346130 |
Yu Qiao1, Yanli Zhao2, Yan Liu3, Ning Ma4, Chuxuan Wang3, Jiaqi Zou5, Zhiyan Liu5, Zhongqiu Zhou5, Dong Han3, Jun He3, Qian Sun3, Yicong Liu3, Changqing Xu6, Zhimin Du5, Hui Huang7.
Abstract
Diabetic cardiomyopathy represents severe heart complications, and is the leading cause of morbidity and mortality among patients with diabetes. Although a few microRNAs (miRNAs) have been implicated in diabetes-related complications, a functional association between miRNAs and cardiac dysfunction in diabetic cardiomyopathy remains to be demonstrated. Our results show that miR-483-3p is upregulated in streptozotocin-induced diabetic mice, and cultured cardiomyocytes mimicking hyperglycemia. Overexpressing miR-483-3p in transgenic mice with diabetes mellitus (DM) exacerbated cardiomyocyte apoptosis by transcriptionally repressing insulin growth factor 1 (IGF1). Therefore, we have uncovered a novel signaling pathway, involving miR-483-3p-IGF1, that promotes myocardial cell apoptosis under high blood-glucose condition. Further, our study indicates that miR-483-3p could be a potential therapeutic target for managing diabetes-associated heart complications.Entities:
Keywords: Apoptosis; Diabetic cardiomyopathy; IGF1; MicroRNA
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Year: 2016 PMID: 27346130 DOI: 10.1016/j.bbrc.2016.06.051
Source DB: PubMed Journal: Biochem Biophys Res Commun ISSN: 0006-291X Impact factor: 3.575