Literature DB >> 27288596

Low-grade endotoxemia, gut permeability and platelet activation in community-acquired pneumonia.

Roberto Cangemi1, Pasquale Pignatelli1, Roberto Carnevale1, Simona Bartimoccia1, Cristina Nocella1, Marco Falcone2, Gloria Taliani3, Francesco Violi4.   

Abstract

OBJECTIVES: Platelet activation seems to be implicated in the cardiovascular events occurring in patients with community-acquired pneumonia (CAP) but the underlying mechanism is still unclear. Aim of the study was to assess the mechanism involved in platelet activation in CAP patients.
METHODS: Two-hundred-seventy-eight consecutive patients hospitalized for CAP were recruited and followed-up until discharge. Hospitalized patients matched for sex, age and comorbidities but without acute infectious diseases were used as controls.
RESULTS: At hospital admission patients disclosed enhanced plasma levels of sP-selectin, a maker of in-vivo platelet activation, serum sNOX2-dp, a marker of NADPH-oxidase activation, serum Lipopolysaccharide (LPS) and serum zonulin, a marker of gut permeability, compared to controls (p < 0.001). Baseline sP-selectin was independently associated to serum LPS, sNOX2-sp and Pneumonia Severity Index score (p < 0.001). Plasma sP-selectin, serum sNOX2-dp, LPS and zonulin coincidentally decreased at hospital discharge (p < 0.001). An in vitro study showed that LPS, at concentration similar to that found in CAP patients, induced sP-selectin release by agonist-activated platelets, a phenomenon that was counteract by treating cells with gp91ds-tat, a specific inhibitor of NOX2.
CONCLUSIONS: CAP patients display enhanced platelet activation, which is related to LPS-mediated NOX2 activation. Enhanced gut permeability seems be implicated in enhancing circulating levels of LPS.
Copyright © 2016 The British Infection Association. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Blood platelets; Endotoxins; NADPH oxidase; P-selectin; Pneumonia

Mesh:

Substances:

Year:  2016        PMID: 27288596     DOI: 10.1016/j.jinf.2016.05.013

Source DB:  PubMed          Journal:  J Infect        ISSN: 0163-4453            Impact factor:   6.072


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