Literature DB >> 27262702

Amyloid- and FDG-PET imaging in amyotrophic lateral sclerosis.

Jordi A Matías-Guiu1, Vanesa Pytel2, María Nieves Cabrera-Martín3, Lucía Galán2, María Valles-Salgado2, Antonio Guerrero2, Teresa Moreno-Ramos2, Jorge Matías-Guiu2, José Luis Carreras3.   

Abstract

PURPOSE: We aimed to study brain metabolism and presence of beta-amyloid deposits using positron emission tomography (PET) in patients with amyotrophic lateral sclerosis (ALS).
METHODS: This prospective cross-sectional study included 18 patients with definite or probable ALS according to the revised El Escorial diagnostic criteria, and 24 healthy controls. Patients underwent neurological and neuropsychological assessments, PET with (18)F-fluorodeoxyglucose (FDG), and amyloid-PET with (18)F-florbetaben.
RESULTS: Patients with ALS showed hypometabolism in the frontal area and hypermetabolism in the cerebellum compared to healthy controls. Four patients (22 %) displayed cognitive impairment and decreased metabolism in the frontal area extending bilaterally to the parietal regions, and increased metabolism in the posterior area of the cerebellum. In patients with no cognitive impairment, metabolism was lower in the left superior frontal gyrus and higher in the anterior and posterior lobes of the cerebellum. In the individual analysis, six patients (35 %) displayed more anterior involvement with hypometabolism affecting the superior frontal, medial, and inferior gyri; six patients (35 %) exhibited a more posterior pattern with hypometabolism in the precentral and postcentral gyri and in the superior and inferior parietal lobules; two patients (11 %) showed a mixed pattern; and three patients (17 %) showed no alterations in brain metabolism. Three (16 %) showed increased (18)F-florbetaben uptake compared to controls.
CONCLUSIONS: We have identified two main patterns of brain metabolism with an association to cognitive status. Only a subgroup of patients showed an increased uptake of the amyloid tracer. Our results suggest that ALS is heterogeneous from a clinical, metabolic, and molecular standpoint.

Entities:  

Keywords:  Alzheimer’s disease; Amyloid; Amyotrophic lateral sclerosis; Brain metabolism; Cognitive impairment; Positron emission tomography

Mesh:

Substances:

Year:  2016        PMID: 27262702     DOI: 10.1007/s00259-016-3434-1

Source DB:  PubMed          Journal:  Eur J Nucl Med Mol Imaging        ISSN: 1619-7070            Impact factor:   9.236


  53 in total

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3.  Validation of the Edinburgh Cognitive and Behavioural Amyotrophic Lateral Sclerosis Screen (ECAS): A cognitive tool for motor disorders.

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4.  Brain hypermetabolism in amyotrophic lateral sclerosis: a FDG PET study in ALS of spinal and bulbar onset.

Authors:  Angelina Cistaro; Maria Consuelo Valentini; Adriano Chiò; Flavio Nobili; Andrea Calvo; Cristina Moglia; Anna Montuschi; Silvia Morbelli; Dario Salmaso; Piercarlo Fania; Giovanna Carrara; Marco Pagani
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9.  The metabolic signature of C9ORF72-related ALS: FDG PET comparison with nonmutated patients.

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Journal:  PLoS One       Date:  2013-02-25       Impact factor: 3.240

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  13 in total

1.  Amyloid- and FDG-PET in sporadic Creutzfeldt-Jakob disease: Correlation with pathological prion protein in neuropathology.

Authors:  Jordi A Matías-Guiu; Carmen Guerrero-Márquez; María Nieves Cabrera-Martín; Ulises Gómez-Pinedo; María Romeral; Diego Mayo; Jesús Porta-Etessam; Teresa Moreno-Ramos; José Luis Carreras; Jorge Matías-Guiu
Journal:  Prion       Date:  2017-05-16       Impact factor: 3.931

Review 2.  Positron emission tomography in amyotrophic lateral sclerosis: Towards targeting of molecular pathological hallmarks.

Authors:  Stefanie M A Willekens; Donatienne Van Weehaeghe; Philip Van Damme; Koen Van Laere
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3.  Testing the diagnostic accuracy of [18F]FDG-PET in discriminating spinal- and bulbar-onset amyotrophic lateral sclerosis.

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5.  Immununochemical Markers of the Amyloid Cascade in the Hippocampus in Motor Neuron Diseases.

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6.  Amyotrophic lateral sclerosis modifies progenitor neural proliferation in adult classic neurogenic brain niches.

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8.  Notch Signalling in the Hippocampus of Patients With Motor Neuron Disease.

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10.  Brain Cortical Complexity Alteration in Amyotrophic Lateral Sclerosis: A Preliminary Fractal Dimensionality Study.

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