Literature DB >> 2725701

Changes in extracellular calcium within the physiological range influence receptor-mediated inositol phosphate responses in brain and tracheal smooth muscle slices.

J G Baird1, E R Chilvers, E D Kennedy, S R Nahorski.   

Abstract

The effect of changes in extracellular calcium concentration ([Ca2+]e) on the incorporation of myo-[2-3H]-inositol into phosphoinositides and agonist-stimulated 3H-inositol phosphates (3H-InsPs) was examined in rat cerebral cortex and bovine tracheal smooth muscle slices. In brain slices, reduction in [Ca2+]e from 2.4 to 1.2 mmol/l resulted in an approximate doubling of the carbachol and noradrenaline-stimulated 3H-InsP response with no effect on the EC50 values. An identical effect of varying [Ca2+]e was observed for carbachol-stimulated 3H-InsP formation in tracheal smooth muscle with a further increase in 3H-InsPs evident at [Ca2+]e 0.6 mmol/l. In this tissue the effect of changes in [Ca2+]e on the incorporation of myo-[2-3H]-inositol into the total phosphoinositide pool directly paralleled the changes in 3H-InsPs except in conditions of no added calcium when 3H-InsP responses were markedly impaired. Additional studies in brain slices using buffer where the added calcium varied between 0 and 2.4 mmol/l, showed that both the carbachol stimulated formation of separate inositol phosphates during short incubation periods and incorporation of myo-[2-3H]-inositol into PtdInsP and PtdInsP2 under basal conditions was maximal at [Ca2+]e 0.3 mmol/l. Omitting Ca2+ from the buffer resulted in maximal labelling of PtdIns but a decrease in PtdInsP and PtdInsP2 labelling (compared with the level at [Ca2+]e 0.3 mmol/l) and a markedly impaired inositol polyphosphate response. Alterations in [Ca2+]e following 3H-inositol labelling but immediately prior to carbachol stimulation did not influence 3H-inositol polyphosphate responses. It is therefore clear that even relatively small changes in [Ca2+]e markedly influence agonist-stimulated 3H-InsP responses in brain and tracheal smooth muscle slices and that these reflect changes in the labelling of substrate inositol lipids.(ABSTRACT TRUNCATED AT 250 WORDS)

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Year:  1989        PMID: 2725701     DOI: 10.1007/bf00173572

Source DB:  PubMed          Journal:  Naunyn Schmiedebergs Arch Pharmacol        ISSN: 0028-1298            Impact factor:   3.000


  21 in total

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Authors:  S R Nahorski; D A Kendall; I Batty
Journal:  Biochem Pharmacol       Date:  1986-08-01       Impact factor: 5.858

2.  Dual pathways in muscarinic receptor stimulation of phosphoinositide hydrolysis.

Authors:  D Gurwitz; M Sokolovsky
Journal:  Biochemistry       Date:  1987-01-27       Impact factor: 3.162

3.  CDP-diglyceride:inositol transferase from rat liver. Purification and properties.

Authors:  T Takenawa; K Egawa
Journal:  J Biol Chem       Date:  1977-08-10       Impact factor: 5.157

4.  Phosphatidylinositol response to cholinergic agonists in airway smooth muscle: relationship to contraction and muscarinic receptor occupancy.

Authors:  B M Grandordy; F M Cuss; A S Sampson; J B Palmer; P J Barnes
Journal:  J Pharmacol Exp Ther       Date:  1986-07       Impact factor: 4.030

5.  Inhibition by Ca2+ of the incorporation of myo-inositol into phosphatidylinositol.

Authors:  K Egawa; T Takenawa; B Sacktor
Journal:  Mol Cell Endocrinol       Date:  1981-01       Impact factor: 4.102

6.  Brain fluid calcium concentration and response to acute hypercalcaemia during development in the rat.

Authors:  H C Jones; R F Keep
Journal:  J Physiol       Date:  1988-08       Impact factor: 5.182

7.  Stimulation of alpha 1-adrenoceptors in rat kidney mediates increased inositol phospholipid hydrolysis.

Authors:  C B Neylon; R J Summers
Journal:  Br J Pharmacol       Date:  1987-06       Impact factor: 8.739

8.  Inhibition of phosphatidylinositol synthesis and the inactivation of calcium entry after prolonged exposure of the blowfly salivary gland to 5-hydroxytryptamine.

Authors:  M J Berridge; J N Fain
Journal:  Biochem J       Date:  1979-01-15       Impact factor: 3.857

9.  Differential receptor occupancy requirements for muscarinic cholinergic stimulation of inositol lipid hydrolysis in brain and in neuroblastomas.

Authors:  S K Fisher; R M Snider
Journal:  Mol Pharmacol       Date:  1987-07       Impact factor: 4.436

10.  [3H]-myo-inositol uptake in rat cortical slices. Identification of Na+-dependent and Na+-independent systems.

Authors:  T C Howerton; C O Rutledge
Journal:  Biochem Pharmacol       Date:  1988-03-01       Impact factor: 5.858

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  4 in total

1.  Receptor-activated calcium entry in exocrine cells does not occur via agonist-sensitive intracellular pools.

Authors:  T J Shuttleworth
Journal:  Biochem J       Date:  1990-03-15       Impact factor: 3.857

2.  Dissociation between phytohaemagglutinin-stimulated generation of inositol phosphates and Ca2+ increase in human mononuclear leucocytes.

Authors:  M C Michel; L J van Tits; G Trenn; J Sykora; O E Brodde
Journal:  Biochem J       Date:  1992-07-01       Impact factor: 3.857

3.  Characterization of agonist-stimulated incorporation of myo-[3H]inositol into inositol phospholipids and [3H]inositol phosphate formation in tracheal smooth muscle.

Authors:  E R Chilvers; P J Barnes; S R Nahorski
Journal:  Biochem J       Date:  1989-09-15       Impact factor: 3.857

4.  Calcium-dependence of histamine- and carbachol-induced inositol phosphate formation in human U373 MG astrocytoma cells: comparison with HeLa cells and brain slices.

Authors:  J A Arias-Montaño; V Berger; J M Young
Journal:  Br J Pharmacol       Date:  1994-02       Impact factor: 8.739

  4 in total

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