Literature DB >> 27170847

Circulating Aldosterone Levels and Disease Severity in Pulmonary Arterial Hypertension.

Zeenat Safdar1, Aishwarya Thakur1, Supriya Singh1, Yingqun Ji1, Danielle Guffey2, Charles G Minard2, Mark L Entman3.   

Abstract

OBJECTIVES: It is not known whether aldosterone levels are associated with increased mortality in patients with pulmonary arterial hypertension (PAH). The primary goal of this study was to determine whether circulating aldosterone levels predict severity of PAH in terms of hemodynamic characteristics and mortality.
METHODS: Patients with stable PAH were enrolled at the Baylor PH program. The plasma levels of aldosterone and BNP were measured. Clinical, hemodynamic, and outcome data was collected by chart review. Mean follow up time from study enrollment was 39 ± 102 months. Cox proportional hazards model was used to assess time to death.
RESULTS: There were 125 PAH patients with plasma aldosterone levels. Median aldosterone level was 9.9 pg/ml (25th-75th percentile: 4.1 pg/ml, 27.1 pg/ml) and median brain natriuretic peptide (BNP) level was 67.5 pg/ml (25th-75th percentile: 31 pg/ml, 225 pg/ml). Aldosterone levels were not significantly associated with BNP levels, six-minute walk distance, Borg dyspnea score, right ventricular systolic pressure, cardiac output and cardiac index. However, the association between aldosterone and right atrial pressure was dependent on mineralocorticoid receptor blocker treatment (Coef. =2.88, 95CI: 1.19, 4.56, p=0.001). By log-rank statistic there was no statistical difference between the survival of patients divided by median aldosterone level (p=0.914). However, there was a significant difference in patient survival between the BNP categories (p<0.001) such that those with high BNP level (>180 pg/mL) had a shorter survival time.
CONCLUSIONS: The aldosterone level was not associated with increased mortality in PAH but was a marker of disease severity.

Entities:  

Keywords:  BNP level; aldosterone; mineralocorticoid receptor antagonist; pulmonary arterial hypertension; right ventricle

Year:  2015        PMID: 27170847      PMCID: PMC4861074          DOI: 10.4172/2161-105X.1000295

Source DB:  PubMed          Journal:  J Pulm Respir Med


  27 in total

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Review 7.  Aldosterone and end-organ damage.

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8.  Plasma brain natriuretic peptide levels increase in proportion to the extent of right ventricular dysfunction in pulmonary hypertension.

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9.  Aldosterone in heart failure: pathophysiology and treatment.

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10.  Clinical significance of brain natriuretic peptide in primary pulmonary hypertension.

Authors:  Hanno H Leuchte; Michael Holzapfel; Rainer A Baumgartner; Isabelle Ding; Claus Neurohr; Michael Vogeser; Tilman Kolbe; Martin Schwaiblmair; Jürgen Behr
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5.  Spironolactone in pulmonary arterial hypertension: results of a cross-over study.

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