Literature DB >> 27140710

Effect of Somatostatin, Ulinastatin and Gabexate on the Treatment of Severe Acute Pancreatitis.

Guiliang Wang1, Yan Liu2, Shu-Feng Zhou3, Ping Qiu4, Linfang Xu4, Ping Wen4, Jianbo Wen4, Xianzhong Xiao5.   

Abstract

OBJECTIVE: The objective of this study is to evaluate the efficacy of somatostatin, ulinastatin and gabexate for the treatment of severe acute pancreatitis.
MATERIALS AND METHODS: A total of 492 patients with severe acute pancreatitis were assigned randomly into the following 4 groups: (1) somatostatin; (2) somatostatin + ulinastatin; (3) somatostatin + gabexate and (4) somatostatin + ulinastatin + gabexate. Acute physiology and chronic health evaluation II scores; clinical parameters including time of abdominal pain and distention extinct; recovering to normality of heart rate and respiration rate; amylase and blood glucose; ratios of efficacy; multiple organ dysfunction syndrome (MODS); mortality; complication; levels of endotoxin; tumor necrosis factor alpha; interleukin-6 (IL-6), IL-8 and IL-10 and side effects were analyzed.
RESULTS: Acute physiology and chronic health evaluation II scores, time of abdominal pain extinct and distention extinct, time of recovering to normality of heart rate, time of recovering to normality of respiration rate and time of recovering to normality of amylase and blood glucose were significantly decreased in the somatostatin + ulinastatin, the somatostatin + gabexate and the somatostatin + ulinastatin + gabexate subgroups compared with the somatostatin subgroup. Ratios of efficacy were significantly improved, whereas ratios of MODS, mortality and complication were significantly decreased in the somatostatin + ulinastatin and the somatostatin + ulinastatin + gabexate subgroups compared with the somatostatin subgroup. Tumor necrosis factor alpha, IL-6 and IL-8 levels on the fourth day after treatment showed significant decrease in the somatostatin + ulinastatin, the somatostatin + gabexate and the somatostatin + ulinastatin + gabexate subgroups compared with the somatostatin subgroup. The IL-10 levels on the fourth day were significantly improved in the somatostatin + ulinastatin, the somatostatin + gabexate and the somatostatin + ulinastatin + gabexate subgroups compared with the somatostatin subgroup.
CONCLUSIONS: Somatostatin is effective for the treatment of acute pancreatitis, ulinastatin demonstrates improvement in therapeutic benefits and gabexate can relieve the clinical symptoms and shorten the course of disease but cannot improve the effective ratio or decrease MODS, mortality and complication.
Copyright © 2016 Southern Society for Clinical Investigation. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Cytokine; Gabexate; Severe acute pancreatitis; Somatostatin; Ulinastatin

Mesh:

Substances:

Year:  2016        PMID: 27140710     DOI: 10.1016/j.amjms.2016.03.013

Source DB:  PubMed          Journal:  Am J Med Sci        ISSN: 0002-9629            Impact factor:   2.378


  19 in total

1.  Predicative values of C-reactive protein for the therapeutic effects of ulinastatin combined with somatostatin in severe acute pancreatitis and for the severity of gastrointestinal failure.

Authors:  Yinjia Wang; Li Li
Journal:  Exp Ther Med       Date:  2018-08-07       Impact factor: 2.447

Review 2.  Peptide-Based Therapeutics for Oncology.

Authors:  Elizaveta Fisher; Kirill Pavlenko; Alexander Vlasov; Galina Ramenskaya
Journal:  Pharmaceut Med       Date:  2019-02

3.  [Mechanism of ulinastatin in reducing lung inflammatory injury in rats with hemorrhagic shock].

Authors:  Ying Chen; Zhipeng Xu; Qi Song; Zhenjie Wang; Zhong Ji; Zhaolei Qiu; Feng Cheng; Hai Jiang
Journal:  Nan Fang Yi Ke Da Xue Xue Bao       Date:  2019-10-30

4.  Tricetin Reduces Inflammation and Acinar Cell Injury in Cerulein-Induced Acute Pancreatitis: The Role of Oxidative Stress-Induced DNA Damage Signaling.

Authors:  Máté Nagy-Pénzes; Zoltán Hajnády; Zsolt Regdon; Máté Á Demény; Katalin Kovács; Tarek El-Hamoly; József Maléth; Péter Hegyi; Csaba Hegedűs; László Virág
Journal:  Biomedicines       Date:  2022-06-10

Review 5.  Pharmacological interventions for acute pancreatitis.

Authors:  Elisabetta Moggia; Rahul Koti; Ajay P Belgaumkar; Federico Fazio; Stephen P Pereira; Brian R Davidson; Kurinchi Selvan Gurusamy
Journal:  Cochrane Database Syst Rev       Date:  2017-04-21

6.  Emodin Alleviates Sodium Taurocholate-Induced Pancreatic Acinar Cell Injury via MicroRNA-30a-5p-Mediated Inhibition of High-Temperature Requirement A/Transforming Growth Factor Beta 1 Inflammatory Signaling.

Authors:  Hong Xiang; Xufeng Tao; Shilin Xia; Jialin Qu; Huiyi Song; Jianjun Liu; Dong Shang
Journal:  Front Immunol       Date:  2017-11-06       Impact factor: 7.561

7.  Associations of MMP-2 and MMP-9 gene polymorphism with ulinastatin efficacy in patients with severe acute pancreatitis.

Authors:  Guo-Dong Zhen; Lian-Bin Zhao; Shan-Shan Wu; Ming-Yu Chen; Zhen-He Li; Sheng-Zhi Zhou; Zhen-Fu Li
Journal:  Biosci Rep       Date:  2017-08-23       Impact factor: 3.840

8.  Henoch-Schönlein purpura with acute pancreatitis: analysis of 13 cases.

Authors:  Qin Zhang; Qi Guo; Ming Gui; Zhenhua Ren; Bo Hu; Ling Lu; Fang Deng
Journal:  BMC Pediatr       Date:  2018-05-11       Impact factor: 2.125

9.  Ulinastatin Inhibits Osteoclastogenesis and Suppresses Ovariectomy-Induced Bone Loss by Downregulating uPAR.

Authors:  Jun-Ming Huang; Ran-Yue Ren; Yuan Bao; Jia-Chao Guo; Wei Xiang; Xing-Zhi Jing; Jia Shi; Guo-Xiang Zhang; Long Li; Yong Tian; Hao Kang; Feng-Jin Guo
Journal:  Front Pharmacol       Date:  2018-09-07       Impact factor: 5.810

10.  Effects of Ulinastatin on Proliferation and Apoptosis of Breast Cancer Cells by Inhibiting the ERK Signaling Pathway.

Authors:  Zeyu Xing; Xin Wang; Jiaqi Liu; Gang Liu; Menglu Zhang; Kexin Feng; Xiang Wang
Journal:  Biomed Res Int       Date:  2021-07-30       Impact factor: 3.411

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.