Literature DB >> 27132877

Genetic Characteristics of Polycythemia Vera and Essential Thrombocythemia in Korean Patients.

Eun-Hyung Yoo1, Kyung-Jin Park2,3, Hong-Hee Won2,3, Jun-Hee Park2,3, Jong-Ho Park2,3, Seung-Tae Lee4, Hee-Jin Kim4, Soo-Mee Bang5, Hyun-Sook Chi6, Chul Won Jung7, Sun-Hee Kim4, Hongseok Yun8, Choong-Hyun Sun8, Inho Park8, Seungmook Lee8, Clarence Lee9, Barry Merriman9, Raymond Luo10, Eileen Hwee Hong Tan10, Keun-Joon Park11, Na-Kyung Yoo11, Jason J Kang9, Jong-Won Kim12.   

Abstract

BACKGROUND: Despite recent advances in the investigation of myeloproliferative neoplasms (MPN), the impact of genetic heterogeneity on its molecular pathogenesis has not been fully elucidated. Thus, in this study, we aim to characterize the genetic complexity in Korean patients with polycythemia vera (PV) and essential thrombocythemia (ET).
METHODS: We conducted association studies using 84 single-nucleotide polymorphisms (SNPs) in 229 patients (96 with PV and 133 with ET) and 170 controls. Further, whole-genome sequencing was performed in six patients (two with JAK2 V617F and four with wild-type JAK2), and putative somatic mutations were validated in a further 69 ET patients. Clinical and laboratory characteristics were also analyzed.
RESULTS: Several germline SNPs and the 46 haplotype were significantly associated with PV and ET. Three somatic mutations in MPDZ, IQCH, and CALR genes were selected and validated. The frequency of the CALR mutation was 58.0% (40/69) in ET patients, who did not carry JAK2/MPL mutations. Moreover, compared with JAK2 V617F-positive patients, those with CALR mutations showed lower hemoglobin and hematocrit levels (P = 0.004 and P = 0.002, respectively), higher platelet counts (P =0.008), and a lower frequency of cytoreductive therapy (P = 0.014).
CONCLUSION: This study was the first comprehensive investigation of the genetic characteristics of Korean patients with PV and ET. We found that somatic mutations and the 46 haplotype contribute to PV and ET pathogenesis in Korean patients.
© 2016 Wiley Periodicals, Inc.

Entities:  

Keywords:  calreticulin; essential thrombocythemia; haplotype; polycythemia vera; whole-genome sequencing

Mesh:

Substances:

Year:  2016        PMID: 27132877      PMCID: PMC6807207          DOI: 10.1002/jcla.21981

Source DB:  PubMed          Journal:  J Clin Lab Anal        ISSN: 0887-8013            Impact factor:   2.352


  36 in total

1.  JAK2V617F mutational frequency in polycythemia vera: 100%, >90%, less?

Authors:  S Verstovsek; R T Silver; N C P Cross; A Tefferi
Journal:  Leukemia       Date:  2006-08-31       Impact factor: 11.528

2.  JAK2 46/1 haplotype is associated with JAK2 V617F-positive myeloproliferative neoplasms in Japanese patients.

Authors:  Mayumi Tanaka; Toshiaki Yujiri; Shunsuke Ito; Naoko Okayama; Toru Takahashi; Kenji Shinohara; Yoichi Azuno; Ryouhei Nawata; Yuji Hinoda; Yukio Tanizawa
Journal:  Int J Hematol       Date:  2013-02-22       Impact factor: 2.490

3.  The JAK2 46/1 haplotype is a risk factor for myeloproliferative neoplasms in Chinese patients.

Authors:  Xinju Zhang; Tingting Hu; Zhiyuan Wu; Zhihua Kang; Weiwei Liu; Ming Guan
Journal:  Int J Hematol       Date:  2012-09-29       Impact factor: 2.490

4.  Analysis of calreticulin mutations in Chinese patients with essential thrombocythemia: clinical implications in diagnosis, prognosis and treatment.

Authors:  R Fu; M Xuan; Y Zhou; T Sun; J Bai; Z Cao; L Zhang; H Li; D Zhang; X Zhang; C Lv; F Xue; X Liu; R Yang; L Zhang
Journal:  Leukemia       Date:  2014-04-15       Impact factor: 11.528

Review 5.  The role of mutations in epigenetic regulators in myeloid malignancies.

Authors:  Alan H Shih; Omar Abdel-Wahab; Jay P Patel; Ross L Levine
Journal:  Nat Rev Cancer       Date:  2012-08-17       Impact factor: 60.716

6.  Novel mutations in the inhibitory adaptor protein LNK drive JAK-STAT signaling in patients with myeloproliferative neoplasms.

Authors:  Stephen T Oh; Erin F Simonds; Carol Jones; Matthew B Hale; Yury Goltsev; Kenneth D Gibbs; Jason D Merker; James L Zehnder; Garry P Nolan; Jason Gotlib
Journal:  Blood       Date:  2010-04-19       Impact factor: 22.113

7.  Integrated genomic analysis illustrates the central role of JAK-STAT pathway activation in myeloproliferative neoplasm pathogenesis.

Authors:  Raajit Rampal; Fatima Al-Shahrour; Omar Abdel-Wahab; Jay P Patel; Jean-Philippe Brunel; Craig H Mermel; Adam J Bass; Jennifer Pretz; Jihae Ahn; Todd Hricik; Outi Kilpivaara; Martha Wadleigh; Lambert Busque; D Gary Gilliland; Todd R Golub; Benjamin L Ebert; Ross L Levine
Journal:  Blood       Date:  2014-04-16       Impact factor: 22.113

8.  MPLW515L is a novel somatic activating mutation in myelofibrosis with myeloid metaplasia.

Authors:  Yana Pikman; Benjamin H Lee; Thomas Mercher; Elizabeth McDowell; Benjamin L Ebert; Maricel Gozo; Adam Cuker; Gerlinde Wernig; Sandra Moore; Ilene Galinsky; Daniel J DeAngelo; Jennifer J Clark; Stephanie J Lee; Todd R Golub; Martha Wadleigh; D Gary Gilliland; Ross L Levine
Journal:  PLoS Med       Date:  2006-07       Impact factor: 11.069

9.  COSMIC: exploring the world's knowledge of somatic mutations in human cancer.

Authors:  Simon A Forbes; David Beare; Prasad Gunasekaran; Kenric Leung; Nidhi Bindal; Harry Boutselakis; Minjie Ding; Sally Bamford; Charlotte Cole; Sari Ward; Chai Yin Kok; Mingming Jia; Tisham De; Jon W Teague; Michael R Stratton; Ultan McDermott; Peter J Campbell
Journal:  Nucleic Acids Res       Date:  2014-10-29       Impact factor: 16.971

10.  JAK2 or CALR mutation status defines subtypes of essential thrombocythemia with substantially different clinical course and outcomes.

Authors:  Elisa Rumi; Daniela Pietra; Virginia Ferretti; Thorsten Klampfl; Ashot S Harutyunyan; Jelena D Milosevic; Nicole C C Them; Tiina Berg; Chiara Elena; Ilaria C Casetti; Chiara Milanesi; Emanuela Sant'antonio; Marta Bellini; Elena Fugazza; Maria C Renna; Emanuela Boveri; Cesare Astori; Cristiana Pascutto; Robert Kralovics; Mario Cazzola
Journal:  Blood       Date:  2013-12-23       Impact factor: 22.113

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