Literature DB >> 27056970

The value of ECG parameters as markers of treatment response in Fabry cardiomyopathy.

Christian Schmied1, Albina Nowak2, Christiane Gruner1, Eric Olinger3, Huguette Debaix3, Andreas Brauchlin1, Michelle Frank1, Saskia Reidt1, Pierre Monney4, Frédéric Barbey5, Dipen Shah6, Mehdi Namdar6.   

Abstract

OBJECTIVE: Best treatment outcomes in Fabry disease (FD) associated cardiomyopathy can be obtained when treatment is started as early as possible. The rationale of this study was to assess the role of ECG changes for identification of cardiac involvement and patients at an earlier stage of the disease more likely deriving a benefit from enzyme replacement therapy (ERT).
METHODS: A retrospective analysis of patient data was performed from an observational, longitudinal, prospective cohort. Treatment response was defined as absence or presence of disease progression, defined as new onset or increase in left ventricular (LV) mass >10%. Demographic, clinical, ECG and echocardiographic parameters at baseline were tested for their value in determining absence or presence of disease progression under ERT at 5-year follow-up.
RESULTS: The study population consisted of a total of 38 patients (25 men, mean age 36±13 years, overall median follow-up duration 6.4±1.2 years). Patients in the progression group (14 men, 4 women) had a longer QRS duration (99±11 ms vs 84±13 ms, p<0.05 for men, 93±9 years vs 81±5 years, p<0.05 for women) and QTc interval (401±15 ms vs 372±10 ms, p<0.005 for men) and a higher amount of ECG abnormalities (86% vs 18%, p<0.005 for men and 100% vs 0%, p<0.005 for women) at the time of ERT initiation. An abnormal baseline ECG was significantly associated with disease progression (sensitivity 94.1%, specificity 88.9%, positive likelihood ratio of 8.47, p<0.005).
CONCLUSIONS: An abnormal ECG at the time of treatment initiation is significantly associated with cardiac disease progression in FD. This effect seems to be independent of age, gender or LV mass at baseline and suggests maximal treatment benefit when ERT is initiated before ECG abnormalities develop. Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/

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Year:  2016        PMID: 27056970     DOI: 10.1136/heartjnl-2015-308897

Source DB:  PubMed          Journal:  Heart        ISSN: 1355-6037            Impact factor:   5.994


  8 in total

1.  Limited effects of long-term enzyme replacement therapy on the cardiac conduction system in Fabry disease.

Authors:  Tomoya Kaneda; Masahiro Takeda; Tetsuro Suematsu; Ryusuke Yamamoto; Mutsuko Takata; Toshinori Higashikata; Hidekazu Ino; Akihiko Tsujibata; Kenshi Hayashi; Noboru Fujino; Masa-Aki Kawashiri
Journal:  J Cardiol Cases       Date:  2018-02-09

2.  Cardiac device implantation in Fabry disease: A retrospective monocentric study.

Authors:  Thomas Sené; Olivier Lidove; Joel Sebbah; Jean-Marc Darondel; Hervé Picard; Laurent Aaron; Olivier Fain; Thierry Zenone; Dominique Joly; Philippe Charron; Jean-Marc Ziza
Journal:  Medicine (Baltimore)       Date:  2016-10       Impact factor: 1.889

3.  Cardiac Phenotype of Prehypertrophic Fabry Disease.

Authors:  Sabrina Nordin; Rebecca Kozor; Shanat Baig; Amna Abdel-Gadir; Katia Medina-Menacho; Stefania Rosmini; Gabriella Captur; Michel Tchan; Tarekegn Geberhiwot; Elaine Murphy; Robin Lachmann; Uma Ramaswami; Nicola C Edwards; Derralynn Hughes; Richard P Steeds; James C Moon
Journal:  Circ Cardiovasc Imaging       Date:  2018-06       Impact factor: 7.792

4.  Heterozygous junctophilin-2 (JPH2) p.(Thr161Lys) is a monogenic cause for HCM with heart failure.

Authors:  Sari U M Vanninen; Krista Leivo; Eija H Seppälä; Katriina Aalto-Setälä; Olli Pitkänen; Piia Suursalmi; Antti-Pekka Annala; Ismo Anttila; Tero-Pekka Alastalo; Samuel Myllykangas; Tiina M Heliö; Juha W Koskenvuo
Journal:  PLoS One       Date:  2018-09-20       Impact factor: 3.240

Review 5.  The effect of enzyme replacement therapy on clinical outcomes in male patients with Fabry disease: A systematic literature review by a European panel of experts.

Authors:  Dominique P Germain; Perry M Elliott; Bruno Falissard; Victor V Fomin; Max J Hilz; Ana Jovanovic; Ilkka Kantola; Aleš Linhart; Renzo Mignani; Mehdi Namdar; Albina Nowak; João-Paulo Oliveira; Maurizio Pieroni; Miguel Viana-Baptista; Christoph Wanner; Marco Spada
Journal:  Mol Genet Metab Rep       Date:  2019-02-06

6.  A randomised controlled trial evaluating arrhythmia burden, risk of sudden cardiac death and stroke in patients with Fabry disease: the role of implantable loop recorders (RaILRoAD) compared with current standard practice.

Authors:  Ravi Vijapurapu; Rebecca Kozor; Derralynn A Hughes; Peter Woolfson; Ana Jovanovic; Patrick Deegan; Rosemary Rusk; Gemma A Figtree; Michel Tchan; David Whalley; Dipak Kotecha; Francisco Leyva; James Moon; Tarekegn Geberhiwot; Richard P Steeds
Journal:  Trials       Date:  2019-05-31       Impact factor: 2.279

7.  The myocardial phenotype of Fabry disease pre-hypertrophy and pre-detectable storage.

Authors:  João B Augusto; Nicolas Johner; Dipen Shah; Sabrina Nordin; Kristopher D Knott; Stefania Rosmini; Clement Lau; Mashael Alfarih; Rebecca Hughes; Andreas Seraphim; Ravi Vijapurapu; Anish Bhuva; Linda Lin; Natalia Ojrzyńska; Tarekegn Geberhiwot; Gabriella Captur; Uma Ramaswami; Richard P Steeds; Rebecca Kozor; Derralynn Hughes; James C Moon; Mehdi Namdar
Journal:  Eur Heart J Cardiovasc Imaging       Date:  2021-06-22       Impact factor: 6.875

8.  Anderson-Fabry disease presenting with atrial fibrillation as earlier sign in a young patient: A case report.

Authors:  Hangyul Kim; Min Gyu Kang; Hyun Woong Park; Jeong-Rang Park; Jin-Yong Hwang; Kyehwan Kim
Journal:  World J Clin Cases       Date:  2021-06-26       Impact factor: 1.337

  8 in total

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