Literature DB >> 27032303

[What's next in glioblastoma treatment: Tumor-targeted or immune-targeted therapies?].

Antoine Schernberg1, Aurélien Marabelle2, Christophe Massard2, Jean-Pierre Armand2, Sarah Dumont2, Eric Deutsch3, Frédéric Dhermain3.   

Abstract

INTRODUCTION: Glioblastoma (GBM) is associated with a poor prognosis. This review will discuss different directions of treatment, mostly regarding immunotherapies and combinatorial approaches. DEVELOPMENT: Standard treatment for newly diagnosed GBM is maximal and safe surgical resection followed by concurrent radiochemotherapy (RCT) based on temozolomide, allowing 14.6 months median survival. Nowadays, no combination with molecular-targeted therapy had significantly improved prognosis. Phases I and II data are emerging, highlighting the potential efficacy of associations with other therapies. Studies have suggested the potential of targeting tumor stem cells, at less partially responsible for resistance to RCT. There is now some evidence that immunotherapy is also relevant for brain tumors. Treatment strategies have mainly explored vaccines strategies, such as the dendritic cell, heat shock protein or EGFRvIII vaccines. Of the work initiated in melanoma, immune checkpoints inhibitors have exhibited stimulating results. Others trials have demonstrated potential of autologous stimulated lymphocytes. Moreover, strong data indicates that radiation therapy has the potential to promote immunogenicity and create a sort of in situ personalized vaccine.
CONCLUSION: These data provide strong evidence to support the potential of associating combinatorial targeted and/or immunotherapeutic regimens in patients with GBM that may change patient outcome.
Copyright © 2016 Société Française du Cancer. Published by Elsevier Masson SAS. All rights reserved.

Entities:  

Keywords:  Glioblastoma multiform; Glioblastome; Immune checkpoints inhibitors; Immunotherapy; Immunothérapie; Radiotherapy; Radiothérapie; Vaccines; Vaccins

Mesh:

Substances:

Year:  2016        PMID: 27032303     DOI: 10.1016/j.bulcan.2016.02.014

Source DB:  PubMed          Journal:  Bull Cancer        ISSN: 0007-4551            Impact factor:   1.276


  3 in total

1.  Sym004-induced EGFR elimination is associated with profound anti-tumor activity in EGFRvIII patient-derived glioblastoma models.

Authors:  Stephen T Keir; Vidyalakshmi Chandramohan; Carlee D Hemphill; Michael M Grandal; Maria Carlsen Melander; Mikkel W Pedersen; Ivan D Horak; Michael Kragh; Annick Desjardins; Henry S Friedman; Darell D Bigner
Journal:  J Neurooncol       Date:  2018-03-21       Impact factor: 4.130

2.  Isoalantolactone inhibits IKKβ kinase activity to interrupt the NF-κB/COX-2-mediated signaling cascade and induces apoptosis regulated by the mitochondrial translocation of cofilin in glioblastoma.

Authors:  Jin-Shan Xing; Xun Wang; Yu-Long Lan; Jia-Cheng Lou; Binbin Ma; Tingzhun Zhu; Hongqiang Zhang; Dongsheng Wang; Zhikuan Yu; Zhongbo Yuan; Xin-Yu Li; Bo Zhang
Journal:  Cancer Med       Date:  2019-02-10       Impact factor: 4.452

3.  Dendritic cells cross-talk with tumour antigen-specific CD8+ T cells, Vγ9γδT cells and Vα24NKT cells in patients with glioblastoma multiforme and in healthy donors.

Authors:  Y Eiraku; H Terunuma; M Yagi; X Deng; A J Nicol; M Nieda
Journal:  Clin Exp Immunol       Date:  2018-09-14       Impact factor: 4.330

  3 in total

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