Literature DB >> 27013231

Development of an angiogenesis animal model featuring brain arteriovenous malformation histological characteristics.

Chrysanthi Papagiannaki1,2, Frédéric Clarençon1,3, Sébastien Ponsonnard1,4, Claude Couquet5, Franck Maizeroi-Eugène6, Damien Bresson7, Catherine Yardin1,8, Charbel Mounayer1,9.   

Abstract

BACKGROUND: Angiogenesis has a key role in the formation and evolution of brain arteriovenous malformations (AVMs). Numerous models have been developed aiming to recreate configuration of brain AVMs.
OBJECTIVE: To develop an animal model sharing the same pathological characteristics as human brain AVMs.
MATERIALS AND METHODS: Ten pigs were divided into two groups. Five animals underwent endovascular left common carotid artery (CCA) and external carotid artery (ECA) occlusion and five animals served as controls. DSA, associated with 3D-rotational angiography, was performed at day 0 and at 3 months in both groups. The volume of the retia was calculated. Vascular endothelial growth factor (VEGF)-A serum levels were measured in both groups at the same time intervals. Finally, the animals were sacrificed at 3 months and the retia were harvested for pathological and immunohistochemistry examinations.
RESULTS: At 3 months, a significantly higher rete volume was seen in group A than in group B (2.92±0.33 mL vs 1.87±0.69 mL, respectively; p=0.016). There was a trend for increased VEGF-A levels in group A at 3 months. In the occlusion group, histological findings showed significant reduction of media thickness and disrupted internal elastic lamina; immunohistochemistry findings showed strong reactivity for VEGF receptors and interleukin 6.
CONCLUSIONS: Unilateral endovascular occlusion of the CCA-ECA results in angiogenesis triggering of the rete mirabile with both significant augmentation of the rete volume and histological evidence of pro-angiogenic stimulation. Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/.

Entities:  

Keywords:  Angiography; Arteriovenous Malformation; Vessel Wall

Mesh:

Substances:

Year:  2016        PMID: 27013231     DOI: 10.1136/neurintsurg-2015-012173

Source DB:  PubMed          Journal:  J Neurointerv Surg        ISSN: 1759-8478            Impact factor:   5.836


  3 in total

1.  Induction of Brain Arteriovenous Malformation Through CRISPR/Cas9-Mediated Somatic Alk1 Gene Mutations in Adult Mice.

Authors:  Wan Zhu; Daniel Saw; Miriam Weiss; Zhengda Sun; Meng Wei; Sonali Shaligram; Sen Wang; Hua Su
Journal:  Transl Stroke Res       Date:  2018-12-03       Impact factor: 6.829

2.  Combination of Alcohol and EVOH as a New Embolic Agent: Midterm Tissue and Inflammatory Effects in a Swine Model.

Authors:  Jean-François Hak; Farouk Tradi; Mickael Bobot; Pauline Brige; Paul Habert; Sophie Chopinet; Aurélie Haffner; Gilles Soulez; Benjamin Guillet; Vincent Vidal
Journal:  Radiol Res Pract       Date:  2020-10-23

3.  Notch signaling pathway is a potential therapeutic target for extracranial vascular malformations.

Authors:  Reema B Davis; Kristy Pahl; Nicholas C Datto; Scott V Smith; Carrie Shawber; Kathleen M Caron; Julie Blatt
Journal:  Sci Rep       Date:  2018-12-20       Impact factor: 4.379

  3 in total

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