Literature DB >> 27003947

Th17 cells: A prognostic marker for MS rebound after natalizumab cessation?

Jürgen Haas1, Katharina Schneider1, Alexander Schwarz1, Mirjam Korporal-Kuhnke1, Simon Faller1, Felipe von Glehn2, Sven Jarius1, Brigitte Wildemann1.   

Abstract

BACKGROUND: Multiple sclerosis (MS) patients are at risk of renewed disease activity after discontinuing natalizumab (NAT) treatment.
OBJECTIVE: Assessing the implication of T helper 17 (Th17) cells in MS reactivation after NAT cessation.
METHODS: We monitored frequencies of Th17 cells and interleukin (IL)-17 cytokine levels in blood samples of 57 MS patients, without, during, and after NAT exposure.
RESULTS: Frequencies of both Th17 cells and, in part, also IL-17 levels, in peripheral blood increased under prolonged NAT therapy, returned to baseline after NAT withdrawal and became almost undetectable in blood samples of individuals who experienced relapses during the wash-out phase.
CONCLUSION: Assessing the Th17-cell/IL-17 axis might help to predict rebound MS activity after NAT withdrawal.

Entities:  

Keywords:  Multiple sclerosis; Th17 effector T cells; natalizumab; rebound effect; wash-out phase

Mesh:

Substances:

Year:  2016        PMID: 27003947     DOI: 10.1177/1352458516640609

Source DB:  PubMed          Journal:  Mult Scler        ISSN: 1352-4585            Impact factor:   6.312


  7 in total

Review 1.  Natalizumab in Multiple Sclerosis: Long-Term Management.

Authors:  Marinella Clerico; Carlo Alberto Artusi; Alessandra Di Liberto; Simona Rolla; Valentina Bardina; Pierangelo Barbero; Stefania Federica De Mercanti; Luca Durelli
Journal:  Int J Mol Sci       Date:  2017-04-29       Impact factor: 5.923

2.  Quantification of γδ T cells and HLA-DR+ NK cells does not predict emergence of new contrast enhancing lesions in MS patients suspending natalizumab treatment.

Authors:  Mindaugas Paužuolis; Torsten Eich; Joachim Burman
Journal:  PLoS One       Date:  2017-06-06       Impact factor: 3.240

Review 3.  Dysregulated MicroRNA Involvement in Multiple Sclerosis by Induction of T Helper 17 Cell Differentiation.

Authors:  Chen Chen; Yifan Zhou; Jingqi Wang; Yaping Yan; Lisheng Peng; Wei Qiu
Journal:  Front Immunol       Date:  2018-06-04       Impact factor: 7.561

4.  Reducing return of disease activity in patients with relapsing multiple sclerosis transitioned from natalizumab to teriflunomide: 12-month interim results of teriflunomide therapy.

Authors:  Stanley L Cohan; Keith Edwards; Lindsay Lucas; Tiffany Gervasi-Follmar; Judy O'Connor; Jessica Siuta; Vineetha Kamath; Lore Garten; Chiayi Chen; James Thomas; Kyle Smoot; Kiren Kresa-Reahl; Kateri J Spinelli
Journal:  Mult Scler J Exp Transl Clin       Date:  2019-01-16

5.  Natalizumab Pharmacokinetics and -Dynamics and Serum Neurofilament in Patients With Multiple Sclerosis.

Authors:  Undine Proschmann; Hernan Inojosa; Katja Akgün; Tjalf Ziemssen
Journal:  Front Neurol       Date:  2021-04-14       Impact factor: 4.003

6.  Th17 cells increase in RRMS as well as in SPMS, whereas various other phenotypes of Th17 increase in RRMS only.

Authors:  S Kalra; C Lowndes; L Durant; R C Strange; A Al-Araji; Clive P Hawkins; S John Curnow
Journal:  Mult Scler J Exp Transl Clin       Date:  2020-01-28

7.  Transcriptional signature of human pro-inflammatory TH17 cells identifies reduced IL10 gene expression in multiple sclerosis.

Authors:  Dan Hu; Samuele Notarbartolo; Tom Croonenborghs; Bonny Patel; Ron Cialic; Tun-Hsiang Yang; Dominik Aschenbrenner; Karin M Andersson; Marco Gattorno; Minh Pham; Pia Kivisakk; Isabelle V Pierre; Youjin Lee; Karun Kiani; Maria Bokarewa; Emily Tjon; Nathalie Pochet; Federica Sallusto; Vijay K Kuchroo; Howard L Weiner
Journal:  Nat Commun       Date:  2017-11-17       Impact factor: 14.919

  7 in total

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