Literature DB >> 26998753

In kidney transplant recipients with BK polyomavirus infection, early BK nephropathy, microvascular inflammation, and serum creatinine are risk factors for graft loss.

M Mohamed1, S Parajuli1, B Muth1, B C Astor1, S E Panzer1, D Mandelbrot1, W Zhong2, A Djamali1.   

Abstract

BACKGROUND: Little information is available on the risk factors for graft loss in kidney transplant recipients with BK polyomavirus (BKPyV) nephropathy (BKVN) in the presence or absence of antibody-mediated rejection (AMR).
METHODS: We examined the risk factors for graft loss in consecutive kidney allograft recipients with biopsy-confirmed BKVN, with or without concomitant AMR.
RESULTS: A total of 1904 kidney transplants were performed at our institution during 2005-2011. Of these, 330 (17.33%) were diagnosed with BKPyV viremia, and 69 were diagnosed with BKVN (3.6%). Eleven patients had a concomitant diagnosis of AMR. Patients with AMR were characterized by significantly higher peak panel-reactive antibody, retransplant rates, and desensitization preconditioning at the time of transplantation, as well as microvascular inflammation (MVI = glomerulitis + peritubular capillaritis), C4d score, and donor-specific antibody at the time of diagnosis (P ≤ 0.01). Treatment with plasma exchange, intravenous immunoglobulin, and cidofovir was more prevalent in this group (P ≤ 0.02). Univariate analyses assessing the risk factors for graft loss in all patients with BKVN, identified an independent association of African-American race, deceased-donor transplantation, serum creatinine (Scr), MVI, and early disease (BKVN within 6 months of transplant) with poor outcomes. Multivariate analyses retained only 3 variables: Scr >2 mg/dL (hazard ratio [HR] = 4.3, 95% confidence interval [CI] 1.9-9.7, P = 0.0004), early BKVN (HR = 2.7, 95% CI 1.3-5.3, P = 0.004), and MVI (HR = 1.8, 95% CI 1.2-2.8, P = 0.008).
CONCLUSIONS: These observations suggest that, in patients with BK infection, early BKVN, Scr >2, and MVI are predictors of poor outcomes. Further studies are needed to determine effective treatment strategies for BKVN, with or without AMR.
© 2016 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

Entities:  

Keywords:  BK nephropathy; BK virus; kidney transplantation; risk factors

Mesh:

Substances:

Year:  2016        PMID: 26998753     DOI: 10.1111/tid.12530

Source DB:  PubMed          Journal:  Transpl Infect Dis        ISSN: 1398-2273            Impact factor:   2.228


  4 in total

1.  BK virus viral load: analysis of the requests received by the microbiology laboratory and clinical involvement of the issued results.

Authors:  Irene Muñoz-Gallego; Noelia Moral; Consuelo Pascual; Yolanda Alonso; Lola Folgueira
Journal:  Eur J Clin Microbiol Infect Dis       Date:  2019-07-12       Impact factor: 3.267

2.  Kidney transplant recipients with polycystic kidney disease have a lower risk of post-transplant BK infection than those with end-stage renal disease due to other causes.

Authors:  Callie Plafkin; Tripti Singh; Brad C Astor; Sandesh Parajuli; Gauri Bhutani; Nasia Safdar; Sarah E Panzer
Journal:  Transpl Infect Dis       Date:  2018-08-30       Impact factor: 2.228

Review 3.  The Role of HLA and KIR Immunogenetics in BK Virus Infection after Kidney Transplantation.

Authors:  Marija Burek Kamenaric; Vanja Ivkovic; Ivana Kovacevic Vojtusek; Renata Zunec
Journal:  Viruses       Date:  2020-12-09       Impact factor: 5.048

Review 4.  BK nephropathy in the native kidneys of patients with organ transplants: Clinical spectrum of BK infection.

Authors:  Darlene Vigil; Nikifor K Konstantinov; Marc Barry; Antonia M Harford; Karen S Servilla; Young Ho Kim; Yijuan Sun; Kavitha Ganta; Antonios H Tzamaloukas
Journal:  World J Transplant       Date:  2016-09-24
  4 in total

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