Literature DB >> 26969815

Exposure-response estimate for lung cancer and asbestosis in a predominantly chrysotile-exposed Chinese factory cohort.

Midori N Courtice1,2, Xiaorong Wang3, Sihao Lin1, Ignatius Tak Sun Yu3, D Wayne Berman4, Eiji Yano5.   

Abstract

BACKGROUND: Cumulative fiber exposures, predominantly chrysotile, were estimated in a Chinese asbestos worker cohort and exposure-response relationships with lung cancer mortality and cumulative incidence of asbestosis were determined.
METHODS: Individual time-dependent cumulative exposures were estimated for 577 asbestos workers, followed prospectively for 37 years. Occupational history and smoking data were obtained from company records and personal interviews; vital status and causes of death were ascertained from death registries and hospital records. Hazard ratios were generated for disease outcomes, with adjustments for smoking and age.
RESULTS: Median cumulative fiber exposure for the cohort was 132.6 fiber-years/ml (IQR 89.3-548.4). Exposure-response relationships were demonstrated for both disease outcomes, with nearly sixfold and threefold increased risks seen at the highest exposure level for lung cancer deaths and asbestosis, respectively.
CONCLUSION: Evidence using quantitative exposure estimates was provided for increased risks of lung cancer mortality and development of asbestosis in a predominantly chrysotile-exposed cohort.
© 2016 Wiley Periodicals, Inc.

Entities:  

Keywords:  Asbestos; asbestosis; cumulative fiber exposure; epidemiology; lung cancer

Mesh:

Substances:

Year:  2016        PMID: 26969815     DOI: 10.1002/ajim.22579

Source DB:  PubMed          Journal:  Am J Ind Med        ISSN: 0271-3586            Impact factor:   2.214


  6 in total

1.  Global DNA hypomethylation has no impact on lung function or serum inflammatory and fibrosis cytokines in asbestos-exposed population.

Authors:  Min Yu; Jianlin Lou; Hailing Xia; Min Zhang; Yixiao Zhang; Junqiang Chen; Xing Zhang; Shibo Ying; Lijin Zhu; Lihong Liu; Guang Jia
Journal:  Int Arch Occup Environ Health       Date:  2017-01-19       Impact factor: 3.015

2.  Lung injury and expression of p53 and p16 in Wistar rats induced by respirable chrysotile fiber dust from four primary areas of China.

Authors:  Yali Zeng; Yan Cui; Ji Ma; Tingting Huo; Faqin Dong; Qingbi Zhang; Jianjun Deng; Xu Zhang; Jie Yang; Yulin Wang
Journal:  Environ Sci Pollut Res Int       Date:  2017-09-29       Impact factor: 4.223

3.  Chromosome nondisjunction during bipolar mitoses of binucleated intermediates promote aneuploidy formation along with multipolar mitoses rather than chromosome loss in micronuclei induced by asbestos.

Authors:  Tianwei Zhang; Lei Lv; Yun Huang; Xiaohui Ren; Qinghua Shi
Journal:  Oncotarget       Date:  2017-02-14

Review 4.  Prevention of Asbestos Exposure in Latin America within a Global Public Health Perspective.

Authors:  Eduardo Algranti; Juan Pablo Ramos-Bonilla; Benedetto Terracini; Vilma S Santana; Pietro Comba; Roberto Pasetto; Agata Mazzeo; Fulvio Cavariani; Andrés Trotta; Daniela Marsili
Journal:  Ann Glob Health       Date:  2019-03-29       Impact factor: 2.462

Review 5.  Adverse health effects of asbestos: solving mysteries regarding asbestos carcinogenicity based on follow-up survey of a Chinese factory.

Authors:  Eiji Yano
Journal:  Environ Health Prev Med       Date:  2018-08-08       Impact factor: 3.674

6.  Relationships of Lower Lung Fibrosis, Pleural Disease, and Lung Mass with Occupational, Household, Neighborhood, and Slate Roof-Dense Area Residential Asbestos Exposure.

Authors:  Dongmug Kang; Yu-Young Kim; Minseung Shin; Min-Su Lee; Hee-Joo Bae; Se-Yeong Kim; Young-Ki Kim
Journal:  Int J Environ Res Public Health       Date:  2018-08-02       Impact factor: 3.390

  6 in total

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