| Literature DB >> 26962945 |
Hayley E Bullen1, Yonggen Jia1, Yoshiki Yamaryo-Botté2, Hugo Bisio1, Ou Zhang3, Natacha Klages Jemelin1, Jean-Baptiste Marq1, Vern Carruthers3, Cyrille Y Botté2, Dominique Soldati-Favre4.
Abstract
The obligate intracellular lifestyle of apicomplexan parasites necessitates an invasive phase underpinned by timely and spatially controlled secretion of apical organelles termed micronemes. In Toxoplasma gondii, extracellular potassium levels and other stimuli trigger a signaling cascade culminating in phosphoinositide-phospholipase C (PLC) activation, which generates the second messengers diacylglycerol (DAG) and IP3 and ultimately results in microneme secretion. Here we show that a delicate balance between DAG and its downstream product, phosphatidic acid (PA), is essential for controlling microneme release. Governing this balance is the apicomplexan-specific DAG-kinase-1, which interconverts PA and DAG, and whose depletion impairs egress and causes parasite death. Additionally, we identify an acylated pleckstrin-homology (PH) domain-containing protein (APH) on the microneme surface that senses PA during microneme secretion and is necessary for microneme exocytosis. As APH is conserved in Apicomplexa, these findings highlight a potentially widely used mechanism in which key lipid mediators regulate microneme exocytosis.Entities:
Keywords: Apicomplexa; Toxoplasma gondii; diacylglycerol kinase; invasion; microneme; phosphatidic acid; phosphatidic acid phosphatase; phosphoinositide-phospholipase C; pleckstrin homology domain; propranolol; regulated exocytosis
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Year: 2016 PMID: 26962945 DOI: 10.1016/j.chom.2016.02.006
Source DB: PubMed Journal: Cell Host Microbe ISSN: 1931-3128 Impact factor: 21.023