Literature DB >> 26959841

Fenofibrate attenuates diabetic nephropathy in experimental diabetic rat's model via suppression of augmented TGF-β1/Smad3 signaling pathway.

Nouf Mohamed Al-Rasheed1, Nawal Mohamed Al-Rasheed1,2, Maha Abdelrahman Al-Amin1, Iman Huesein Hasan1, Hanaa Najeeb Al-Ajmi1, Raeesa Ahmed Mohammad3, Hala Aboulfotooh Attia1,4.   

Abstract

CONTEXT: Fibrates, the ligands of peroxisome profileferator-activated receptor-α have been shown to have a renal protective action in diabetic nephropathy (DN).
OBJECTIVE: This study aimed to elucidate the effect of fenofibrate on renal transforming growth factor-β1 (TGF-β1) and Smad3 in Streptozotocin (STZ)-induced DN.
METHODS: Diabetes was induced in rats by a single intraperitoneal injection of streptozotocin (55 mg/kg). Diabetic rats were given fenofibrate (100 mg/kg, p.o.). After 12 weeks, diabetic nephropathy biomarkers were assessed. The mRNA expression of collage I and III, TGF-β1 and Smad3 and were detected by RT-PCR.
RESULTS: Fenofibrate reduced significantly serum creatinine, kidney/body weight ratio, serum albumin excretion Collage I & III, TGF-β1 and Smad3 mRNA expression.
CONCLUSIONS: Our results give further insights into the mechanisms underlying the protective role of fenofibrate in DN, suggesting that interference with TGF-β1/Smad3 signaling pathway may be a useful therapeutic approach to prevent DN.

Entities:  

Keywords:  Animals; Smad3; TGF-β1; antibody; diabetic nephropathy; drugs; fenofibrate

Mesh:

Substances:

Year:  2016        PMID: 26959841     DOI: 10.3109/13813455.2016.1164186

Source DB:  PubMed          Journal:  Arch Physiol Biochem        ISSN: 1381-3455            Impact factor:   4.076


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